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Paper Details

Sex-dependent compensatory mechanisms preserve blood pressure homeostasis in prostacyclin receptor-deficient mice.
J Clin Invest
1
2021
ANP, I prostanoid receptor, Ipr, Ipr-/-, Ldlr, Ldlr-/-, PGE2, PGI2, atherogenesis, atrial natriuretic peptide, estrogen, hyperlipidemia, hyperlipidemic, hypertension, low-density lipoprotein receptor, mPGES-1, mPges, mPges-1, mPges-1-/-, men, mice, microsomal prostaglandin E synthase 1, prostacyclin, prostacyclin receptor, prostaglandin E, prostaglandin E2
Author NameAffiliation
Nicholas F LahensInstitute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania
Emanuela RicciottiInstitute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania
Kyle BittingerChildren's Hospital of Philadelphia
Gregory R GrantInstitute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania
Gregory R GrantUniversity of Pennsylvania
Garret A FitzGeraldInstitute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania
Garret A FitzGeraldInstitute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania
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Release 1.2