Skip to Main Content

Paper Details

EZHIP/CXorf67 mimics K27M mutated oncohistones and functions as an intrinsic inhibitor of PRC2 function in aggressive posterior fossa ependymoma.
Neuro Oncol
91
2019
C, CXorf67, EZH Inhibitory Protein, EZH2, EZH2 methyltransferase, EZHIP, H3K27, H3K27M, H3K27me3, K27M mutated histones, PFA, PFA ependymoma, PFA tumors, PRC2, PRC2 target genes, SET domain, Su(var)3-9, aggressive posterior fossa ependymoma, children, chromosome X open reading frame 67, diffuse midline gliomas, enhancer-of-zeste, ependymoma, histone H3, infants, lysine, oncohistones, polycomb repressive complex 2, trithorax, tumors, zeste
Author NameAffiliation
Robert B RussellHeidelberg University Biochemistry Center
Robert B RussellHeidelberg University
David W EllisonSt Jude Children's Research Hospital
Stefan M PfisterGerman Cancer Research Center (DKFZ)
Stefan M PfisterUniversity Hospital
Stefan M PfisterHopp Children's Cancer Center
Stefan M PfisterGerman Cancer Research Center (DKFZ)
Stefan M PfisterHopp Children's Cancer Center
Stefan M PfisterUniversity Hospital
Kristian W PajtlerGerman Cancer Research Center (DKFZ)
Kristian W PajtlerUniversity Hospital
Kristian W PajtlerHopp Children's Cancer Center
Kristian W PajtlerGerman Cancer Research Center (DKFZ)
Kristian W PajtlerHopp Children's Cancer Center
Kristian W PajtlerUniversity Hospital
Marcel KoolGerman Cancer Research Center (DKFZ)
Marcel KoolHopp Children's Cancer Center
Marcel KoolGerman Cancer Research Center (DKFZ)
Marcel KoolHopp Children's Cancer Center
  • 1 - 19

Datasets

Release 1.2