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Paper Details

Inborn errors of OAS-RNase L in SARS-CoV-2-related multisystem inflammatory syndrome in children.
Science
52
2023
-deficient cells, 2-, 2-5A, COVID-19, Inborn errors of OAS, MAVS, MDA5, MDA5 or RIG-I deficiency, MIS-C, Monocytic cell lines, Multisystem inflammatory syndrome, OAS, OAS-RNase L deficiencies, OAS1, OAS1, OAS2, or RNase L deficiencies, OAS1-deficient but not RNase L-deficient cells, OAS2, RIG-I, RNase L, RNase L-deficient cells, SARS-CoV-2, autosomal recessive deficiencies of , , or, children, coronavirus, dsRNA, mitochondrial antiviral-signaling protein, mitochondrial antiviral-signaling protein (MAVS) deficiency, mononuclear phagocytes, multisystem inflammatory syndrome, patients, primary myeloid cells, severe acute respiratory syndrome coronavirus, single, single-stranded RNA
Author NameAffiliation
Aurélie BisiauxInstitut Pasteur, Paris City University, CNRS UMR 0
Hans-Heinrich HoffmannThe Rockefeller University
Kaya BilguvarDepartments of Neurosurgery and Genetics and Yale Center for Genome Analysis, Yale School of Medicine
Kaya BilguvarAcibadem Mehmet Ali Aydinlar University
Shrikant ManeYale University School of Medicine
Tom Maniatis
Richard P LiftonYale University School of Medicine
Richard P LiftonThe Rockefeller University
Richard P LiftonYale University School of Medicine
Richard P LiftonThe Rockefeller University
Charles M RiceThe Rockefeller University
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