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Paper Details

A non-coding variant linked to metabolic obesity with normal weight affects actin remodelling in subcutaneous adipocytes.
Nat Metab
5
2023
2q24, COBLL1, COBLL1 gene, Cobll1, MONW, POU domain, actin, differentiating subcutaneous adipocytes, effector gene, effector gene COBLL1, glucose, impaired glucose tolerance, insulin, insulin resistance, insulin-sensitive adipocytes, intronic single-, metabolic obesity, metabolically obese normal weight, mouse, non-coding variant, rs6712203, subcutaneous adipocytes, target genes, transcription factor 2, type 2 diabetes
Author NameAffiliation
Nezar AbdennurInstitute for Medical Engineering and Sciences, Massachusetts Institute of Technology
Anne E CarpenterBroad Institute of MIT and Harvard
Anne E CarpenterBroad Institute of MIT and Harvard
Cecilia M LindgrenBroad Institute of MIT and Harvard
Cecilia M LindgrenBig Data Institute at the Li Ka Shing Centre for Health Information and Discovery, University of Oxford
Melina ClaussnitzerBroad Institute of MIT and Harvard
Melina ClaussnitzerNovo Nordisk Foundation Center for Genomic Mechanisms of Disease, Broad Institute of MIT and Harvard
Melina ClaussnitzerDiabetes Unit and Center for Genomic Medicine, Massachusetts General Hospital
Melina ClaussnitzerHarvard Medical School
Melina ClaussnitzerBroad Institute of MIT and Harvard
Melina ClaussnitzerNovo Nordisk Foundation Center for Genomic Mechanisms of Disease, Broad Institute of MIT and Harvard
Melina ClaussnitzerDiabetes Unit and Center for Genomic Medicine, Massachusetts General Hospital
Melina ClaussnitzerHarvard Medical School
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