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Paper Details

Knock-in of the <i>Wt1</i> R394W mutation causes MDS and cooperates with <i>Flt3/ITD</i> to drive aggressive myeloid neoplasms in mice.
Oncotarget
6
2018
FLT3, FMS-like tyrosine kinase 3, Flt3, Flt3 +/ITD, ITD, MDS, MPN, NK, NK-AML, R394W, WT1, WT1 gene, WT1 mutations, WT1-mutant AML, Wilms tumor, Wilms tumor 1, Wt1, Wt1 +/R394W, Wt1 R394W mutation, Wt1 mutation, hematologic, hematologic malignancies, hematopoietic progenitor cells, human, leukemia, malignancies, mice, mouse, myelodysplastic syndrome, myeloid neoplasms, myeloid progenitor cells, normal, oncogene, patients, tumor, tyrosine, zinc
Author NameAffiliation
Li LiThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University School of Medicine
Li LiThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University School of Medicine
Donald SmallThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University School of Medicine
Donald SmallJohns Hopkins University School of Medicine
Donald SmallThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University School of Medicine
Donald SmallJohns Hopkins University School of Medicine
David M LoebAlbert Einstein College of Medicine
Patrick BrownThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University School of Medicine
Patrick BrownJohns Hopkins University School of Medicine
Patrick BrownThe Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins, Johns Hopkins University School of Medicine
Patrick BrownJohns Hopkins University School of Medicine
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Release 1.2