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Paper Details

Toll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus Infection.
mBio
322
2015
ARDS, Coronavirus, ISG, MERS-CoV, Middle East respiratory syndrome coronavirus, MyD88, SARS, SARS-CoV, SARS-CoV disease, Severe Acute Respiratory Syndrome Coronavirus, TLR, TLR3, TLR4, TLRs, TRAM, TRIF, TRIF adaptor protein, TRIF(-/-), Toll-Like Receptor 3, Toll-like receptors, acute respiratory distress syndrome, coronavirus, host cell, human, inflammatory cell, influenza virus infections, interferon-stimulated gene, mice, mouse, neutrophils, patients, severe, severe acute respiratory syndrome coronavirus, wild-type mice
Author NameAffiliation
Allison L ToturaUniversity of North Carolina-Chapel Hill
Alan C WhitmoreUniversity of North Carolina-Chapel Hill
Sudhakar AgnihothramUniversity of North Carolina-Chapel Hill
Alexandra Sch??ferUniversity of North Carolina-Chapel Hill
Michael G KatzeUniversity of Washington
Michael G KatzeUniversity of Washington
Ralph S Baric
Ralph S Baric
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