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Paper Details

Oncogenic <i>KRAS</i> Recruits an Expansive Transcriptional Network through Mutant p53 to Drive Pancreatic Cancer Metastasis.
Cancer Discov
58
2021
-, -catenin, CREB1, FOXA1, KRAS, Mutant p53, Oncogenic KRAS, Oncogenic KRAS mutations, PDAC, PDAC tumors, Pancreatic Cancer, Pancreatic ductal adenocarcinoma, TP53, TP53 tumor suppressor, WNT, cancers, catenin, human, mutant p53, oncogene, oncogenic KRAS, patients, transcription factor, tumor, tumor suppressor gene
Author NameAffiliation
Florencia McAllisterThe University of Texas MD Anderson Cancer Center
Florencia McAllisterThe University of Texas MD Anderson Cancer Center
Christopher A BristowInstitute for Applied Cancer Science, The University of Texas MD Anderson Cancer Center
Christopher A BristowInstitute for Applied Cancer Science, The University of Texas MD Anderson Cancer Center
Timothy P HeffernanInstitute for Applied Cancer Science, The University of Texas MD Anderson Cancer Center
Anirban MaitraSheikh Ahmed Pancreatic Cancer Research Center, The University of Texas MD Anderson Cancer Center
Amanda R WasylishenThe University of Texas MD Anderson Cancer Center
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