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Paper Details

Uncompensated mitochondrial oxidative stress underlies heart failure in an iPSC-derived model of congenital heart disease.
Cell Stem Cell
15
2022
-CM, HF, HLHS, Hypoplastic left heart syndrome, TUDCA, congenital heart disease, heart failure, iPSC, iPSC-CM, mitochondrial dysfunction, patient, patients, sildenafil
Author NameAffiliation
Gloria S PryhuberDepartments of Pediatrics and Environmental Medicine University of Rochester Medical Center Rochester
Gloria S PryhuberDepartments of Pediatrics and Environmental Medicine University of Rochester Medical Center Rochester
George A PorterAab Cardiovascular Research Institute, University of Rochester Medical Center
Bruce J AronowCincinnati Children's Hospital Medical Center, University of Cincinnati, University of Cincinnati School of Medicine
Bruce J AronowCincinnati Children's Hospital Medical Center, University of Cincinnati, University of Cincinnati School of Medicine
Cecilia W LoUniversity of Pittsburgh
Cecilia W LoUniversity of Pittsburgh
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Datasets

Release 1.2