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Paper Details

Persistent activation of autophagy in kidney tubular cells promotes renal interstitial fibrosis during unilateral ureteral obstruction.
174
2016
ACTA2, ACTA2/-, Atg7, FGF2, FN1, PT, Renal fibrosis, TGFB1, Tubular atrophy, UUO, VIM, autophagy-related 7, collagen fibrils, end-stage renal disease, fibroblast growth factor 2, fibroblasts, fibronectin 1, fibrosis, interstitial inflammation, kidney, kidney proximal tubules, kidney tubular cells, macrophage, mice, nephron, primary proximal tubular cells, renal fibrosis, renal interstitial fibrosis, smooth muscle actin, transforming growth factor 1, tubular cell, unilateral ureteral obstruction, vimentin
Author NameAffiliation
Shuang HuangUniversity of Florida College of Medicine
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