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Paper Details

STAT3 gain-of-function mutations associated with autoimmune lymphoproliferative syndrome like disease deregulate lymphocyte apoptosis and can be targeted by BH3 mimetic compounds.
Clin Immunol
40
2017
-, ABT-737, ALPS, ALPS-like disease, Autoimmune lymphoproliferative syndrome, BCL2 family proteins, BCL2-, BH3, FAS, STAT3, STAT3-mutant cells, anti-apoptotic BCL2 proteins, autoimmune lymphoproliferative syndrome like disease, children, lymphocyte, mutant cells, patients
Author NameAffiliation
Michael GombertHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University Dusseldorf
Arndt BorkhardtHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University Dusseldorf
Arndt BorkhardtHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University Dusseldorf
Ute FischerHematology and Clinical Immunology, University Children's Hospital, Heinrich-Heine-University Dusseldorf
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Release 1.2