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Paper Details

Increased soluble urokinase plasminogen activator levels modulate monocyte function to promote atherosclerosis.
J Clin Invest
18
2022
CCL2, PLAUR gene, Pcsk9, People, Soluble, atherosclerosis, atherosclerotic, atherosclerotic plaques, cardiovascular, cholesterol, coronary artery, coronary disease, kidney disease, macrophage, mice, monocyte, participants, rs4760, suPAR, suPARTg, suPARTg monocytes, urokinase plasminogen activator receptor
Author NameAffiliation
George HindyLund University
George HindyBroad Institute of MIT and Harvard
George HindyQatar University College of Medicine
Nona SotoodehniaCardiovascular Health Research Unit, University of Washington
Nona SotoodehniaCardiovascular Health Research Unit, University of Washington
James S PankowUniversity of Minnesota
Christian ErikstrupAarhus University Hospital
Ole Birger PedersenZealand University Hospital
S??ren BrunakNovo Nordisk Foundation Center for Protein Research, University of Copenhagen
S??ren BrunakNovo Nordisk Foundation Center for Protein Research, University of Copenhagen
Sisse R OstrowskiCopenhagen University Hospital
Luca A LottaRegeneron Pharmaceuticals Inc.
Olle MelanderLund University
Marju Orho-MelanderLund University
Cristen J WillerUniversity of Michigan ann arbor
Cristen J WillerUniversity of Michigan ann arbor
Susan R HeckbertUniversity of Washington
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