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Paper Details

Epilepsy in a mouse model of GNB1 encephalopathy arises from altered potassium (GIRK) channel signaling and is alleviated by a GIRK inhibitor.
Front Cell Neurosci
2
2023
Ca, Cultured mutant cortical neurons, ETX, Epilepsy, G, G protein-coupled potassium, G proteins, GIRK, GIRK channels, GNB1, GNB1 encephalopathy, T, cultured neurons, developmental delay, encephalopathy, epilepsy, ethosuximide, generalized seizures, mice, mouse, neurodevelopmental disorder, neuronal, oocytes, potassium
Author NameAffiliation
David B GoldsteinInstitute for Genomic Medicine, Columbia University Irving Medical Center
David B GoldsteinColumbia University Irving Medical Center
David B GoldsteinInstitute for Genomic Medicine, Columbia University Irving Medical Center
David B GoldsteinColumbia University Irving Medical Center
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Release 1.2