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Author Details

Matthew J Christopher
Washington University School of Medicine
2001
20
15
PMIDPaper TitleJournal TitlePublished Year
36563336Flotetuzumab and other T-cell immunotherapies upregulate MHC class II expression on acute myeloid leukemia cells.Blood2023
34670359Tumor suppressor function of <i>WT1</i> in acute promyelocytic leukemia.Haematologica2022
33434062Biology of Disease Relapse in Myeloid Disease: Implication for Strategies to Prevent and Treat Disease Relapse After Stem-Cell Transplantation.J Clin Oncol2021
33704937Genome Sequencing as an Alternative to Cytogenetic Analysis in Myeloid Cancers.N Engl J Med2021
34845035Immunosuppression and outcomes in adult patients with de novo acute myeloid leukemia with normal karyotypes.Proc Natl Acad Sci U S A2021
29618788DNMT3A<sup>R882</sup>-associated hypomethylation patterns are maintained in primary AML xenografts, but not in the DNMT3A<sup>R882C</sup> OCI-AML3 leukemia cell line.Blood Cancer J2018
30207916Mutation Clearance after Transplantation for Myelodysplastic Syndrome.N Engl J Med2018
30380364Immune Escape of Relapsed AML Cells after Allogeneic Transplantation.N Engl J Med2018
27058228A genomic analysis of Philadelphia chromosome-negative AML arising in patients with CML.Blood Cancer J2016
27181063Comprehensive genomic analysis reveals FLT3 activation and a therapeutic strategy for a patient with relapsed adult B-lymphoblastic leukemia.Exp Hematol2016
26305651Association Between Mutation Clearance After Induction Therapy and Outcomes in Acute Myeloid Leukemia.JAMA2015
24518205G-CSF regulates hematopoietic stem cell activity, in part, through activation of Toll-like receptor signaling.Leukemia2014
25092143Primary acute myeloid leukemia cells with IDH1 or IDH2 mutations respond to a DOT1L inhibitor in vitro.Leukemia2014
23434756CXCL12 in early mesenchymal progenitors is required for haematopoietic stem-cell maintenance.Nature2013
21282380Expression of the G-CSF receptor in monocytic cells is sufficient to mediate hematopoietic progenitor mobilization by G-CSF in mice.J Exp Med2011
19141863Suppression of CXCL12 production by bone marrow osteoblasts is a common and critical pathway for cytokine-induced mobilization.Blood2009
18597629Granulocyte colony-stimulating factor induces osteoblast apoptosis and inhibits osteoblast differentiation.J Bone Miner Res2008
17133093Regulation of neutrophil homeostasis.Curr Opin Hematol2007
16037394G-CSF potently inhibits osteoblast activity and CXCL12 mRNA expression in the bone marrow.Blood2005
11536304Methylation and mutational analysis of p27(kip1) in prostate carcinoma.Prostate2001
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Collaborators

Washington University School of Medicine
Co-authored papers 13
Washington University School of Medicine
Co-authored papers 9
Washington University School of Medicine
Co-authored papers 8
Washington University School of Medicine
Co-authored papers 8
Washington University School of Medicine in St. Louis
Co-authored papers 6
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Co-authored papers 6
McDonnell Genome Institute, Washington University School of Medicine
Co-authored papers 6
Washington University School of Medicine
Co-authored papers 6
McDonnell Genome Institute, Washington University School of Medicine
Co-authored papers 6
Washington University School of Medicine in St. Louis
Co-authored papers 6
McDonnell Genome Institute, Washington University in St. Louis School of Medicine
Co-authored papers 5
Washington University School of Medicine.
Co-authored papers 5
Washington University School of Medicine.
Co-authored papers 5
St. Jude Children's Research Hospital
Co-authored papers 4
Massachusetts General Hospital
Co-authored papers 4
Washington University
Co-authored papers 3
The Ohio State University
Co-authored papers 3
The Ohio State University
Co-authored papers 3
Washington University
Co-authored papers 3
St. Jude Children's Research Hospital
Co-authored papers 2
National Institutes of Health Office of the Director
Co-authored papers 2
Co-authored papers 2
The Ohio State University
Co-authored papers 2
Centene Center for Health Transformation, Centene Corporation
Co-authored papers 2
McDonnell Genome Institute, Washington University School of Medicine
Co-authored papers 2
McDonnell Genome Institute, Washington University School of Medicine
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Washington University in St. Louis School of Medicine
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Washington University School of Medicine
Co-authored papers 2
Prostate Cancer Research Center, Tampere University and Tays Cancer Center
Co-authored papers 1
Ontario Institute for Cancer Research
Co-authored papers 1