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Author Details

John D Mountz
University of Alabama at Birmingham and Birmingham VA Medical Center.
1978
224
58
Jake Y. Chen (CM4AI)
PMIDPaper TitleJournal TitlePublished Year
36682593Interrelation of T cell cytokines and autoantibodies in systemic lupus erythematosus: A cross-sectional study.Clin Immunol2023
37981105Increased development of T-bet<sup>+</sup>CD11c<sup>+</sup> B cells predisposes to lupus in females: Analysis in BXD2 mouse and genetic crosses.Clin Immunol2023
36682593Interrelation of T cell cytokines and autoantibodies in systemic lupus erythematosus: A cross-sectional study.Clin Immunol2023
37981105Increased development of T-bet<sup>+</sup>CD11c<sup>+</sup> B cells predisposes to lupus in females: Analysis in BXD2 mouse and genetic crosses.Clin Immunol2023
34725967Host Genetics But Not Commensal Microbiota Determines the Initial Development of Systemic Autoimmune Disease in BXD2 Mice.Arthritis Rheumatol2022
36165181IL-4-Induced Quiescence of Resting Naive B Cells Is Disrupted in Systemic Lupus Erythematosus.J Immunol2022
35436902Lupus nephritis correlates with B cell interferon-β, anti-Smith, and anti-DNA: a retrospective study.Arthritis Res Ther2022
36189576IL-4 receptor blockade is a global repressor of naïve B cell development and responses in a dupilumab-treated patient.Clin Immunol2022
34725967Host Genetics But Not Commensal Microbiota Determines the Initial Development of Systemic Autoimmune Disease in BXD2 Mice.Arthritis Rheumatol2022
35436902Lupus nephritis correlates with B cell interferon-β, anti-Smith, and anti-DNA: a retrospective study.Arthritis Res Ther2022
36165181IL-4-Induced Quiescence of Resting Naive B Cells Is Disrupted in Systemic Lupus Erythematosus.J Immunol2022
36189576IL-4 receptor blockade is a global repressor of naïve B cell development and responses in a dupilumab-treated patient.Clin Immunol2022
32554431IL-23 Promotes a Coordinated B Cell Germinal Center Program for Class-Switch Recombination to IgG2b in BXD2 Mice.J Immunol2020
32554431IL-23 Promotes a Coordinated B Cell Germinal Center Program for Class-Switch Recombination to IgG2b in BXD2 Mice.J Immunol2020
30833421Dysregulation of T Follicular Helper Cells in Lupus.J Immunol2019
31631359Autoreactive B cells in SLE, villains or innocent bystanders?Immunol Rev2019
30833421Dysregulation of T Follicular Helper Cells in Lupus.J Immunol2019
31631359Autoreactive B cells in SLE, villains or innocent bystanders?Immunol Rev2019
29439293Editorial: STATus of STAT3 in Psoriatic Arthritis.Arthritis Rheumatol2018
30201809Cutting Edge: Intracellular IFN-β and Distinct Type I IFN Expression Patterns in Circulating Systemic Lupus Erythematosus B Cells.J Immunol2018
29526711Unmasking Fucosylation: from Cell Adhesion to Immune System Regulation and Diseases.Cell Chem Biol2018
29439293Editorial: STATus of STAT3 in Psoriatic Arthritis.Arthritis Rheumatol2018
29466691Role of production of type I interferons by B cells in the mechanisms and pathogenesis of systemic lupus erythematosus.Discov Med2018
30201809Cutting Edge: Intracellular IFN-β and Distinct Type I IFN Expression Patterns in Circulating Systemic Lupus Erythematosus B Cells.J Immunol2018
29526711Unmasking Fucosylation: from Cell Adhesion to Immune System Regulation and Diseases.Cell Chem Biol2018
29466691Role of production of type I interferons by B cells in the mechanisms and pathogenesis of systemic lupus erythematosus.Discov Med2018
28904124Cutting Edge: Endogenous IFN-β Regulates Survival and Development of Transitional B Cells.J Immunol2017
28904124Cutting Edge: Endogenous IFN-β Regulates Survival and Development of Transitional B Cells.J Immunol2017
25879791Odanacatib, A Cathepsin K-Specific Inhibitor, Inhibits Inflammation and Bone Loss Caused by Periodontal Diseases.J Periodontol2015
25888644General Approach for Tetramer-Based Identification of Autoantigen-Reactive B Cells: Characterization of La- and snRNP-Reactive B Cells in Autoimmune BXD2 Mice.J Immunol2015
25879791Odanacatib, A Cathepsin K-Specific Inhibitor, Inhibits Inflammation and Bone Loss Caused by Periodontal Diseases.J Periodontol2015
25836298Identification of follicular T helper cells in tissue sections.Methods Mol Biol2015
26098211Interferon-induced mechanosensing defects impede apoptotic cell clearance in lupus.J Clin Invest2015
26098211Interferon-induced mechanosensing defects impede apoptotic cell clearance in lupus.J Clin Invest2015
25888644General Approach for Tetramer-Based Identification of Autoantigen-Reactive B Cells: Characterization of La- and snRNP-Reactive B Cells in Autoimmune BXD2 Mice.J Immunol2015
25836298Identification of follicular T helper cells in tissue sections.Methods Mol Biol2015
24909430Interleukin-21 promotes germinal center reaction by skewing the follicular regulatory T cell to follicular helper T cell balance in autoimmune BXD2 mice.Arthritis Rheumatol2014
24909430Interleukin-21 promotes germinal center reaction by skewing the follicular regulatory T cell to follicular helper T cell balance in autoimmune BXD2 mice.Arthritis Rheumatol2014
24838610Inhibition of fucosylation reshapes inflammatory macrophages and suppresses type II collagen-induced arthritis.Arthritis Rheumatol2014
25093822Dysregulated cytokine production by dendritic cells modulates B cell responses in the NZM2410 mouse model of lupus.PLoS One2014
25001511IL-23 promotes TCR-mediated negative selection of thymocytes through the upregulation of IL-23 receptor and RORγt.Nat Commun2014
25001511IL-23 promotes TCR-mediated negative selection of thymocytes through the upregulation of IL-23 receptor and RORγt.Nat Commun2014
25093822Dysregulated cytokine production by dendritic cells modulates B cell responses in the NZM2410 mouse model of lupus.PLoS One2014
24838610Inhibition of fucosylation reshapes inflammatory macrophages and suppresses type II collagen-induced arthritis.Arthritis Rheumatol2014
23543760Cutting Edge: defective follicular exclusion of apoptotic antigens due to marginal zone macrophage defects in autoimmune BXD2 mice.J Immunol2013
25309946The Dynamic Duo-Inflammatory M1 macrophages and Th17 cells in Rheumatic Diseases.J Orthop Rheumatol2013
25392765Increased vitamin D is associated with decline of naïve, but accumulation of effector, CD8 T cells during early aging.Adv Aging Res2013
23858031IL-17RA is essential for optimal localization of follicular Th cells in the germinal center light zone to promote autoantibody-producing B cells.J Immunol2013
23818250Extension of the germinal center stage of B cell development promotes autoantibodies in BXD2 mice.Arthritis Rheum2013
23818173Death receptor 5-targeted depletion of interleukin-23-producing macrophages, Th17, and Th1/17 associated with defective tyrosine phosphatase in mice and patients with rheumatoid arthritis.Arthritis Rheum2013
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Collaborators

Division of Clinical Immunology and Rheumatology, University of Alabama at Birmingham
Co-authored papers 99
University of Texas MD Anderson Cancer Center
Co-authored papers 67
University of Michigan Medical School
Co-authored papers 41
University of Alabama at Birmingham
Co-authored papers 11
Anhui Institute of Innovative Drugs, School of Pharmacy, Anhui Medical University
Co-authored papers 10
Mayo Clinic
Co-authored papers 9
University of Tennessee Health Science Center
Co-authored papers 7
University of Alabama at Birmingham Birmingham
Co-authored papers 6
University of Alabama at Birmingham
Co-authored papers 6
University of Alabama at Birmingham
Co-authored papers 3
Co-authored papers 3
Co-authored papers 3
University of California davis
Co-authored papers 3
Co-authored papers 2
University of Alabama at Birmingham
Co-authored papers 2
Center for Systems Neurogenetics of Addiction at The Jackson Laboratory
Co-authored papers 2
Merck & Co., Inc.
Co-authored papers 2
University of Alabama
Co-authored papers 2
College Station
Co-authored papers 2
The Jackson Laboratory Cancer Center
Co-authored papers 1
Sanford Burnham Prebys Medical Discovery Institute
Co-authored papers 1
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University of Florida College of Medicine.
Co-authored papers 1
National Cancer Institute, National Institutes of Health
Co-authored papers 1
University of Virginia
Co-authored papers 1
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Center for Cancer Research, National Cancer Institute
Co-authored papers 1
University of Wisconsin
Co-authored papers 1
Hospital for Special Surgery and Weill Cornell Medical College
Co-authored papers 1