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Author Details

Daniel Zingg
Netherlands Cancer Institute
2001
23
15
PMIDPaper TitleJournal TitlePublished Year
37642941MYC is a clinically significant driver of mTOR inhibitor resistance in breast cancer.J Exp Med2023
37642941MYC is a clinically significant driver of mTOR inhibitor resistance in breast cancer.J Exp Med2023
36050473Publisher Correction: Truncated FGFR2 is a clinically actionable oncogene in multiple cancers.Nature2022
36050473Publisher Correction: Truncated FGFR2 is a clinically actionable oncogene in multiple cancers.Nature2022
32117586Response of metastatic mouse invasive lobular carcinoma to mTOR inhibition is partly mediated by the adaptive immune system.Oncoimmunology2020
32117586Response of metastatic mouse invasive lobular carcinoma to mTOR inhibition is partly mediated by the adaptive immune system.Oncoimmunology2020
31039140SMAD signaling promotes melanoma metastasis independently of phenotype switching.J Clin Invest2019
31039140SMAD signaling promotes melanoma metastasis independently of phenotype switching.J Clin Invest2019
30951662Yin Yang 1 Orchestrates a Metabolic Program Required for Both Neural Crest Development and Melanoma Formation.Cell Stem Cell2019
30951662Yin Yang 1 Orchestrates a Metabolic Program Required for Both Neural Crest Development and Melanoma Formation.Cell Stem Cell2019
28737469Social and economic burden of recurrent urinary tract infections and quality of life: a patient web-based study (GESPRIT).Expert Rev Pharmacoecon Outcomes Res2018
28898550Rare, yet relevant tumor cells - A new twist to melanoma cell plasticity.Pigment Cell Melanoma Res2018
28737469Social and economic burden of recurrent urinary tract infections and quality of life: a patient web-based study (GESPRIT).Expert Rev Pharmacoecon Outcomes Res2018
30008323EZH2-Mediated Primary Cilium Deconstruction Drives Metastatic Melanoma Formation.Cancer Cell2018
30008323EZH2-Mediated Primary Cilium Deconstruction Drives Metastatic Melanoma Formation.Cancer Cell2018
28898550Rare, yet relevant tumor cells - A new twist to melanoma cell plasticity.Pigment Cell Melanoma Res2018
28746871The Histone Methyltransferase Ezh2 Controls Mechanisms of Adaptive Resistance to Tumor Immunotherapy.Cell Rep2017
28746871The Histone Methyltransferase Ezh2 Controls Mechanisms of Adaptive Resistance to Tumor Immunotherapy.Cell Rep2017
27903862Improved cancer immunotherapy by a CD25-mimobody conferring selectivity to human interleukin-2.Sci Transl Med2016
27903862Improved cancer immunotherapy by a CD25-mimobody conferring selectivity to human interleukin-2.Sci Transl Med2016
25609585The epigenetic modifier EZH2 controls melanoma growth and metastasis through silencing of distinct tumour suppressors.Nat Commun2015
25885555Methylation-dependent SOX9 expression mediates invasion in human melanoma cells and is a negative prognostic factor in advanced melanoma.Genome Biol2015
25629959Antagonistic cross-regulation between Sox9 and Sox10 controls an anti-tumorigenic program in melanoma.PLoS Genet2015
25609585The epigenetic modifier EZH2 controls melanoma growth and metastasis through silencing of distinct tumour suppressors.Nat Commun2015
25885555Methylation-dependent SOX9 expression mediates invasion in human melanoma cells and is a negative prognostic factor in advanced melanoma.Genome Biol2015
25629959Antagonistic cross-regulation between Sox9 and Sox10 controls an anti-tumorigenic program in melanoma.PLoS Genet2015
22772081Sox10 promotes the formation and maintenance of giant congenital naevi and melanoma.Nat Cell Biol2012
22772081Sox10 promotes the formation and maintenance of giant congenital naevi and melanoma.Nat Cell Biol2012
21239582Sulfate-driven elemental sparing is regulated at the transcriptional and posttranscriptional levels in a filamentous cyanobacterium.J Bacteriol2011
21239582Sulfate-driven elemental sparing is regulated at the transcriptional and posttranscriptional levels in a filamentous cyanobacterium.J Bacteriol2011
18086371Responsiveness of fibrocytes to toll-like receptor danger signals.Immunobiology2007
18086371Responsiveness of fibrocytes to toll-like receptor danger signals.Immunobiology2007
15064712Degradation of PKB/Akt protein by inhibition of the VEGF receptor/mTOR pathway in endothelial cells.Oncogene2004
27137923Abstracts from the 8th Annual Meeting of the Scientific Association of Swiss Radiation Oncology (SASRO).Strahlenther Onkol2004
15289348Differential activation of the phosphatidylinositol 3'-kinase/Akt survival pathway by ionizing radiation in tumor and primary endothelial cells.Cancer Res2004
15064712Degradation of PKB/Akt protein by inhibition of the VEGF receptor/mTOR pathway in endothelial cells.Oncogene2004
14751504Novel radiosensitizers for locally advanced epithelial tumors: inhibition of the PI3K/Akt survival pathway in tumor cells and in tumor-associated endothelial cells as a novel treatment strategy?Int J Radiat Oncol Biol Phys2004
27137923Abstracts from the 8th Annual Meeting of the Scientific Association of Swiss Radiation Oncology (SASRO).Strahlenther Onkol2004
15289348Differential activation of the phosphatidylinositol 3'-kinase/Akt survival pathway by ionizing radiation in tumor and primary endothelial cells.Cancer Res2004
14751504Novel radiosensitizers for locally advanced epithelial tumors: inhibition of the PI3K/Akt survival pathway in tumor cells and in tumor-associated endothelial cells as a novel treatment strategy?Int J Radiat Oncol Biol Phys2004
12678723Signal transduction inhibitors as radiosensitizers.Curr Med Chem Anticancer Agents2002
12678723Signal transduction inhibitors as radiosensitizers.Curr Med Chem Anticancer Agents2002
11157943One of two hemN genes in Bradyrhizobium japonicum is functional during anaerobic growth and in symbiosis.J Bacteriol2001
11719451The phosphatidylinositide 3'-kinase/Akt survival pathway is a target for the anticancer and radiosensitizing agent PKC412, an inhibitor of protein kinase C.Cancer Res2001
11157943One of two hemN genes in Bradyrhizobium japonicum is functional during anaerobic growth and in symbiosis.J Bacteriol2001
11719451The phosphatidylinositide 3'-kinase/Akt survival pathway is a target for the anticancer and radiosensitizing agent PKC412, an inhibitor of protein kinase C.Cancer Res2001
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Collaborators

Institute of Anatomy, University of Zurich
Co-authored papers 10
University Hospital Zurich
Co-authored papers 7
University Hospital Zurich
Co-authored papers 6
University Hospital of Zurich
Co-authored papers 5
Institute of Anatomy, University of Zurich
Co-authored papers 4
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Co-authored papers 4
University of Zurich
Co-authored papers 4
Oncode Institute, Netherlands Cancer Institute
Co-authored papers 3
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Co-authored papers 3
University Medical Centre Mannheim, University of Heidelberg
Co-authored papers 3
Institut fur Biochemie, Friedrich-Alexander-Universitat Erlangen-Nurnberg
Co-authored papers 2
University Hospital Basel, University of Basel
Co-authored papers 2
Zurich University Hospital
Co-authored papers 2
University Hospital Zurich
Co-authored papers 2
University Hospital Zurich
Co-authored papers 2
Northwestern University
Co-authored papers 1
Foundation Medicine
Co-authored papers 1
Incyte Research Institute
Co-authored papers 1
Moores Cancer Center, University of California
Co-authored papers 1
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Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 1
University Hospital Zurich, University of Zurich
Co-authored papers 1
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Johns Hopkins School of Medicine, National Cancer Institute, Princeton University, University of Colorado Anschutz Medical Campus, University of Pittsburgh, University of Washington
Co-authored papers 1
Robert Wood Johnson Medical School, The State University of New Jersey
Co-authored papers 1
Hartwig Medical Foundation
Co-authored papers 1
Biostatistics and Prevention Institute, University of Zurich
Co-authored papers 1
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 1
University of Southern California
Co-authored papers 1