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Author Details
Full Name
Jonathan Kay
Affiliation
University College London
ORCID
Career Start Year
2013
Papers
15
H Index
11
Expertise
CM4AI Collaborator
PMID
Paper Title
Journal Title
Published Year
29662167
Sequencing of prostate cancers identifies new cancer genes, routes of progression and drug targets.
Nat Genet
2018
30073682
Immunohistochemical biomarker validation in highly selective needle biopsy microarrays derived from mpMRI-characterized prostates.
Prostate
2018
28945760
Appraising the relevance of DNA copy number loss and gain in prostate cancer using whole genome DNA sequence data.
PLoS Genet
2017
28292578
Corrigendum to "Integration of Copy Number and Transcriptomics Provides Risk Stratification in Prostate Cancer: A Discovery and Validation Cohort Study" [EBioMedicine 2 (9) (2015) 1133-1144].
EBioMedicine
2017
27578825
Whole blood mRNA in prostate cancer reveals a four-gene androgen regulated panel.
Endocr Relat Cancer
2016
26572708
The Early Effects of Rapid Androgen Deprivation on Human Prostate Cancer.
Eur Urol
2016
27732966
HNF1B variants associate with promoter methylation and regulate gene networks activated in prostate and ovarian cancer.
Oncotarget
2016
25560400
HES5 silencing is an early and recurrent change in prostate tumourigenesis.
Endocr Relat Cancer
2015
26501111
Integration of copy number and transcriptomics provides risk stratification in prostate cancer: A discovery and validation cohort study.
EBioMedicine
2015
26035357
Epigenetic and oncogenic regulation of SLC16A7 (MCT2) results in protein over-expression, impacting on signalling and cellular phenotypes in prostate cancer.
Oncotarget
2015
26018901
Corrigendum: analysis of the genetic phylogeny of multifocal prostate cancer identifies multiple independent clonal expansions in neoplastic and morphologically normal prostate tissue.
Nat Genet
2015
25730763
Analysis of the genetic phylogeny of multifocal prostate cancer identifies multiple independent clonal expansions in neoplastic and morphologically normal prostate tissue.
Nat Genet
2015
24240687
N-acetyl-L-aspartyl-L-glutamate peptidase-like 2 is overexpressed in cancer and promotes a pro-migratory and pro-metastatic phenotype.
Oncogene
2014
22806573
Method for sampling tissue for research which preserves pathological data in radical prostatectomy.
Prostate
2013
23880827
Peroxiredoxin-3 is overexpressed in prostate cancer and promotes cancer cell survival by protecting cells from oxidative stress.
Br J Cancer
2013
1 - 15 of 15
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