Author details

Full Name

Steven P Jones

Affiliation

ORCID

Career Start Year

1998

Papers

H Index

Expertise

CM4AI Collaborator

PMID	Paper Title	Journal Title	Published Year
33979180	The Sweet Smell of Progress With Hyaluronan and Heart Failure.	Hypertension	2021
33253217	Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarction.	PLoS ONE	2020
32442025	Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarction.	American Journal of Physiology - Heart and Circulatory Physiology	2020
31310161	Physiological Biomimetic Culture System for Pig and Human Heart Slices.	Circ Res	2019
31639389	Metabolic regulation of Kv channels and cardiac repolarization by KvÎ²2 subunits.	Journal of Molecular and Cellular Cardiology	2019
31300413	RDH10 function is necessary for spontaneous fetal mouth movement that facilitates palate shelf elevation.	DMM Disease Models and Mechanisms	2019
29420208	RNA Editing: Unexplored Opportunities in the Cardiovascular System.	Circulation Research	2018
29885625	Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overload.	Redox Biol	2018
29627295	Deficiency of aldose reductase exacerbates early pressure overload-induced cardiac dysfunction and autophagy in mice.	Journal of Molecular and Cellular Cardiology	2018
29351451	Guidelines for experimental models of myocardial ischemia and infarction.	Am J Physiol Heart Circ Physiol	2018
29483092	Regulates Igf2bp2 Translation in Cardiomyocytes.	Circulation Research	2018
28238121	Leukocyte iNOS is required for inflammation and pathological remodeling in ischemic heart failure.	Basic Research in Cardiology	2017
28860122	Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac Growth.	Circulation	2017
28596166	I'll Have the Rigor, but Hold the Mortis.	Circulation Research	2017
28626081	Why publish in the ?	American Journal of Physiology - Heart and Circulatory Physiology	2017
28706006	Integration of flux measurements to resolve changes in anabolic and catabolic metabolism in cardiac myocytes.	Biochemical Journal	2017
29242383	Correction: Integration of flux measurements to resolve changes in anabolic and catabolic metabolism in cardiac myocytes.	Biochemical Journal	2017
28299467	Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophy.	Basic Research in Cardiology	2017
27536657	A New Method to Stabilize C-Kit Expression in Reparative Cardiac Mesenchymal Cells.	Frontiers in Cell and Developmental Biology	2016
27591224	TNF receptor signaling inhibits cardiomyogenic differentiation of cardiac stem cells and promotes a neuroadrenergic-like fate.	American Journal of Physiology - Heart and Circulatory Physiology	2016
26527687	E2F1 Transcription Factor Regulates O-linked N-acetylglucosamine (O-GlcNAc) Transferase and O-GlcNAcase Expression.	Journal of Biological Chemistry	2015
25499773	The NHLBI-sponsored Consortium for preclinicAl assESsment of cARdioprotective therapies (CAESAR): a new paradigm for rigorous, accurate, and reproducible evaluation of putative infarct-sparing interventions in mice, rabbits, and pigs.	Circulation Research	2015
25627821	High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation.	Biochemical Journal	2015
26565625	O-GlcNAcylation Negatively Regulates Cardiomyogenic Fate in Adult Mouse Cardiac Mesenchymal Stromal Cells.	PLoS ONE	2015
26358111	Recent Developments in Heart Failure.	Circulation Research	2015
26342068	Induction of activating transcription factor 3 limits survival following infarct-induced heart failure in mice.	American Journal of Physiology - Heart and Circulatory Physiology	2015
25183011	MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expression.	Journal of Biological Chemistry	2014
24287310	O-GlcNAc and the cardiovascular system.	Pharmacology and Therapeutics	2014
24186210	Cardiomyocyte Ogt is essential for postnatal viability.	Am J Physiol Heart Circ Physiol	2014
25206341	Endothelial dysfunction as a nexus for endothelial cell-cardiomyocyte miscommunication.	Frontiers in Physiology	2014
24762972	Metabolomic analysis of pressure-overloaded and infarcted mouse hearts.	Circ Heart Fail	2014
23335157	Protein O-GlcNAcylation is a novel cytoprotective signal in cardiac stem cells.	Stem Cells	2013
24367585	High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.	PLoS ONE	2013
23395176	O-GlcNAc signaling entrains the circadian clock by inhibiting BMAL1/CLOCK ubiquitination.		2013
23308291	Reduced cardiac fructose 2,6 bisphosphate increases hypertrophy and decreases glycolysis following aortic constriction.	PLoS ONE	2013
23421427	PDGF-mediated autophagy regulates vascular smooth muscle cell phenotype and resistance to oxidative stress.	Biochem J	2013
23092951	Standardized bioenergetic profiling of adult mouse cardiomyocytes.	Physiological Genomics	2012
22844439	The COX-2/PGI2 receptor axis plays an obligatory role in mediating the cardioprotection conferred by the late phase of ischemic preconditioning.	PLoS ONE	2012
22408028	O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophy.	American Journal of Physiology - Heart and Circulatory Physiology	2012
21873502	Cardiac overexpression of 8-oxoguanine DNA glycosylase 1 protects mitochondrial DNA and reduces cardiac fibrosis following transaortic constriction.	American Journal of Physiology - Heart and Circulatory Physiology	2011
20798965	Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes.	Amino Acids	2011
21147079	Bioenergetic function in cardiovascular cells: the importance of the reserve capacity and its biological regulation.	Chem Biol Interact	2011
21275902	Responses of hypertrophied myocytes to reactive species: implications for glycolysis and electrophile metabolism.	Biochemical Journal	2011
20651294	O-GlcNAc signaling in the cardiovascular system.	Circulation Research	2010
20876116	O-linked Î²-N-acetylglucosamine transferase is indispensable in the failing heart.	Proceedings of the National Academy of Sciences of the United States of America	2010
20215409	TRO40303, a new cardioprotective compound, inhibits mitochondrial permeability transition.	Journal of Pharmacology and Experimental Therapeutics	2010
19023128	Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury.	Circulation Research	2009
19213962	AMP-dependent protein kinase activators: not just for diabetes?	Circulation Research	2009
19734355	O-GlcNAc signaling attenuates ER stress-induced cardiomyocyte death.	American Journal of Physiology - Heart and Circulatory Physiology	2009
18539296	Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transition.	Journal of Molecular and Cellular Cardiology	2008