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Author Details
Full Name
Clare Johnson
Affiliation
College of Life Sciences, University of Dundee
ORCID
Career Start Year
2013
Papers
12
H Index
12
Expertise
CM4AI Collaborator
Trey Ideker (CM4AI)
PMID
Paper Title
Journal Title
Published Year
33060197
Comparative host-coronavirus protein interaction networks reveal pan-viral disease mechanisms.
Science
2020
30017913
The MALDI-TOF E2/E3 Ligase Assay as Universal Tool for Drug Discovery in the Ubiquitin Pathway.
Cell Chem Biol
2018
28368371
CUL-2<sup>LRR-1</sup> and UBXN-3 drive replisome disassembly during DNA replication termination and mitosis.
Nat Cell Biol
2017
28414322
Parkin-phosphoubiquitin complex reveals cryptic ubiquitin-binding site required for RBR ligase activity.
Nat Struct Mol Biol
2017
27477512
UBQLN2 Mediates Autophagy-Independent Protein Aggregate Clearance by the Proteasome.
Cell
2016
25505247
Biophysical studies on interactions and assembly of full-size E3 ubiquitin ligase: suppressor of cytokine signaling 2 (SOCS2)-elongin BC-cullin 5-ring box protein 2 (RBX2).
J Biol Chem
2015
26286618
Characterisation of the Cullin-3 mutation that causes a severe form of familial hypertension and hyperkalaemia.
EMBO Mol Med
2015
26116755
Binding to serine 65-phosphorylated ubiquitin primes Parkin for optimal PINK1-dependent phosphorylation and activation.
EMBO Rep
2015
25752573
K29-selective ubiquitin binding domain reveals structural basis of specificity and heterotypic nature of k29 polyubiquitin.
Mol Cell
2015
24647965
Phosphorylation of Parkin at Serine65 is essential for activation: elaboration of a Miro1 substrate-based assay of Parkin E3 ligase activity.
Open Biol
2014
24885147
VAPB/ALS8 interacts with FFAT-like proteins including the p97 cofactor FAF1 and the ASNA1 ATPase.
BMC Biol
2014
23387299
The CUL3-KLHL3 E3 ligase complex mutated in Gordon's hypertension syndrome interacts with and ubiquitylates WNK isoforms: disease-causing mutations in KLHL3 and WNK4 disrupt interaction.
Biochem J
2013
1 - 12 of 12
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