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Author Details

Suzanne M J Fleiszig
University of California berkeley
1992
116
43
PMIDPaper TitleJournal TitlePublished Year
36758675Contact lens-induced corneal parainflammation involving Ly6G+ cell infiltration requires IL-17A and γδ T cells.Ocul Surf2023
37589460Intracellular replication of <i>Pseudomonas aeruginosa</i> in epithelial cells requires suppression of the caspase-4 inflammasome.mSphere2023
37585189TRPA1 and TPRV1 Ion Channels Are Required for Contact Lens-Induced Corneal Parainflammation and Can Modulate Levels of Resident Corneal Immune Cells.Invest Ophthalmol Vis Sci2023
36824932Intracellular replication of Pseudomonas aeruginosa in epithelial cells requires suppression of the caspase-4 inflammasome.bioRxiv2023
35130333Exotoxin S secreted by internalized Pseudomonas aeruginosa delays lytic host cell death.PLoS Pathog2022
35739166Topical antibiotics reduce CD11c+ cell numbers in the healthy murine cornea and modulate their response to contact lens wear.Sci Rep2022
36374039Pseudomonas aeruginosa Can Diversify after Host Cell Invasion to Establish Multiple Intracellular Niches.mBio2022
34332149Human tear fluid modulates the Pseudomonas aeruginosa transcriptome to alter antibiotic susceptibility.Ocul Surf2021
34569661Nerve-associated transient receptor potential ion channels can contribute to intrinsic resistance to bacterial adhesion in vivo.FASEB J2021
34044593Microbial Science Research in the Post-COVID Environment.mBio2021
34166700Quantification of relative neurite tortuosity using Fourier transforms.J Neurosci Methods2021
32470039Resistance of the murine cornea to bacterial colonization during experimental dry eye.PLoS One2020
31756497Contact lens-related corneal infection: Intrinsic resistance and its compromise.Prog Retin Eye Res2020
30439473A novel murine model for contact lens wear reveals clandestine IL-1R dependent corneal parainflammation and susceptibility to microbial keratitis upon inoculation with Pseudomonas aeruginosa.Ocul Surf2019
31819296Infectious corneal ulceration: a proposal for neglected tropical disease status.Bull World Health Organ2019
31431558Type IV Pili Can Mediate Bacterial Motility within Epithelial Cells.mBio2019
31511582DMBT1 inhibition of Pseudomonas aeruginosa twitching motility involves its N-glycosylation and cannot be conferred by the Scavenger Receptor Cysteine-Rich bacteria-binding peptide domain.Sci Rep2019
30343040Impact of topical corticosteroid pretreatment on susceptibility of the injured murine cornea to Pseudomonas aeruginosa colonization and infection.Exp Eye Res2019
29717012The Impact of ExoS on <i>Pseudomonas aeruginosa</i> Internalization by Epithelial Cells Is Independent of <i>fleQ</i> and Correlates with Bistability of Type Three Secretion System Gene Expression.mBio2018
29896179IL-1R and MyD88 Contribute to the Absence of a Bacterial Microbiome on the Healthy Murine Cornea.Front Microbiol2018
29770353Quantification of Bacterial Twitching Motility in Dense Colonies Using Transmitted Light Microscopy and Computational Image Analysis.Bio Protoc2018
29518189Epithelial cell lysates induce ExoS expression and secretion by Pseudomonas aeruginosa.FEMS Microbiol Lett2018
27670247Human Tear Fluid Reduces Culturability of Contact Lens-Associated Pseudomonas aeruginosa Biofilms but Induces Expression of the Virulence-Associated Type III Secretion System.Ocul Surf2017
28489917Mucosal fluid glycoprotein DMBT1 suppresses twitching motility and virulence of the opportunistic pathogen Pseudomonas aeruginosa.PLoS Pathog2017
28223334Corneal surface glycosylation is modulated by IL-1R and <i>Pseudomonas aeruginosa</i> challenge but is insufficient for inhibiting bacterial binding.FASEB J2017
29062042Contributions of MyD88-dependent receptors and CD11c-positive cells to corneal epithelial barrier function against Pseudomonas aeruginosa.Sci Rep2017
28898291Correction: Mucosal fluid glycoprotein DMBT1 suppresses twitching motility and virulence of the opportunistic pathogen Pseudomonas aeruginosa.PLoS Pathog2017
27375592Pseudomonas aeruginosa Outer Membrane Vesicles Triggered by Human Mucosal Fluid and Lysozyme Can Prime Host Tissue Surfaces for Bacterial Adhesion.Front Microbiol2016
25714715Pseudomonas aeruginosa-induced bleb-niche formation in epithelial cells is independent of actinomyosin contraction and enhanced by loss of cystic fibrosis transmembrane-conductance regulator osmoregulatory function.mBio2015
25955639Pseudomonas aeruginosa Survival at Posterior Contact Lens Surfaces after Daily Wear.Optom Vis Sci2015
25667266The importance of the Pseudomonas aeruginosa type III secretion system in epithelium traversal depends upon conditions of host susceptibility.Infect Immun2015
24658300Cornea and ocular surface disease: application of cutting-edge optometric research.Optom Vis Sci2014
24761794Cytotoxic clinical isolates of Pseudomonas aeruginosa identified during the Steroids for Corneal Ulcers Trial show elevated resistance to fluoroquinolones.BMC Ophthalmol2014
233475713. Ocular surface health with contact lens wear.Cont Lens Anterior Eye2013
24058462Pseudomonas aeruginosa utilizes the type III secreted toxin ExoS to avoid acidified compartments within epithelial cells.PLoS One2013
23601656Why does the healthy cornea resist Pseudomonas aeruginosa infection?Am J Ophthalmol2013
23762428Surfactant protein D contributes to ocular defense against Pseudomonas aeruginosa in a murine model of dry eye disease.PLoS One2013
23469087MicroRNA-762 is upregulated in human corneal epithelial cells in response to tear fluid and Pseudomonas aeruginosa antigens and negatively regulates the expression of host defense genes encoding RNase7 and ST2.PLoS One2013
23411878Association between cytotoxic and invasive Pseudomonas aeruginosa and clinical outcomes in bacterial keratitis.JAMA Ophthalmol2013
23266587Microbial keratitis: could contact lens material affect disease pathogenesis?Eye Contact Lens2013
233475724. Contemporary research in contact lens care.Cont Lens Anterior Eye2013
22167094Traversal of multilayered corneal epithelia by cytotoxic Pseudomonas aeruginosa requires the phospholipase domain of exoU.Invest Ophthalmol Vis Sci2012
22981600Translocon-independent intracellular replication by Pseudomonas aeruginosa requires the ADP-ribosylation domain of ExoS.Microbes Infect2012
23006328Cytokeratins mediate epithelial innate defense through their antimicrobial properties.J Clin Invest2012
21051692Factors impacting corneal epithelial barrier function against Pseudomonas aeruginosa traversal.Invest Ophthalmol Vis Sci2011
22355527Modulation of epithelial immunity by mucosal fluid.Sci Rep2011
21843628Adenylate cyclase activity of Pseudomonas aeruginosa ExoY can mediate bleb-niche formation in epithelial cells and contributes to virulence.Microb Pathog2011
219011513D quantitative imaging of unprocessed live tissue reveals epithelial defense against bacterial adhesion and subsequent traversal requires MyD88.PLoS One2011
21723562Dynamics of Pseudomonas aeruginosa association with anionic hydrogel surfaces in the presence of aqueous divalent-cation salts.J Colloid Interface Sci2011
21498762Dynamics of flagellum- and pilus-mediated association of Pseudomonas aeruginosa with contact lens surfaces.Appl Environ Microbiol2011
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Collaborators

Fiona Stapleton
University of New South Wales
Co-authored papers 4
Thomas M Lietman
Francis I Proctor Foundation, University of California san francisco
Co-authored papers 3
Muthiah Srinivasan
Aravind Eye Hospital
Co-authored papers 3
Nisha R Acharya
F.I. Proctor Foundation, University of California san francisco
Co-authored papers 3
Prajna Lalitha
Aravind Eye Hospital and Postgraduate Institute of Ophthalmology
Co-authored papers 3
Joanne N Engel
University of California san francisco
Co-authored papers 2
Stephen D McLeod
University of California san francisco
Co-authored papers 2
Carolyn R Bertozzi
Stanford University
Co-authored papers 2
Michael E Zegans
Geisel School of Medicine at Dartmouth
Co-authored papers 2
Grischa Chandy
Co-authored papers 1
Arturo Casadevall
Johns Hopkins Bloomberg School of Public Health
Co-authored papers 1
Eduardo C Alfonso
Bascom Palmer Eye Institute, University of Miami
Co-authored papers 1
Elmer Y Tu
University of Illinois at Chicago
Co-authored papers 1
Rasik B Vajpayee
Vision Eye Institute
Co-authored papers 1
Hugh R Taylor
The University of Melbourne
Co-authored papers 1
Sonal S Tuli
University of Florida Shands Hospital
Co-authored papers 1
John K G Dart
Moorfields Eye Hospital NHS Foundation Trust, The UCL Institute of Ophthalmology
Co-authored papers 1
Prashant Garg
Shantilal Shanghvi Cornea Institute, LV Prasad Eye Institute
Co-authored papers 1
Namrata Sharma
All India Institute of Medical Sciences(AIIMS)
Co-authored papers 1
David C Gritz
Johns Hopkins Wilmer Eye Institute (T.M.L., Johns Hopkins University
Co-authored papers 1
Darlene Miller
Bascom Palmer Eye Institute, University of Miami Miller School of Medicine
Co-authored papers 1
Savitri Sharma
L V Prasad Eye Institute
Co-authored papers 1
Radhika Tandon
All India Institute of Medical Sciences
Co-authored papers 1
Michael S Gilmore
Harvard Medical School, USA Infectious Disease Institute (P.J.M.B.
Co-authored papers 1
Lawson Ung
Harvard Medical School
Co-authored papers 1
Todd P Margolis
Washington University School of Medicine
Co-authored papers 1
Stephanie L Watson
The University of Sydney, Save Sight Institute
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Gerami D Seitzman
F.I. Proctor Foundation, University of California san francisco
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Release 1.2