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Author Details

Hongchuan Li
Frederick National Laboratory for Cancer Research
2008
29
14
PMIDPaper TitleJournal TitlePublished Year
37976160Abundant binary promoter switches in lineage-determining transcription factors indicate a digital component of cell fate determination.Cell Rep2023
37801092The KIR2DL1 intermediate upstream element participates in gene activation.Immunogenetics2023
36230818HPV16 E7 Nucleotide Variants Found in Cancer-Free Subjects Affect E7 Protein Expression and Transformation.Cancers (Basel)2022
35858344Genetic variation that determines <i>TAPBP</i> expression levels associates with the course of malaria in an HLA allotype-dependent manner.Proc Natl Acad Sci U S A2022
32784501Genetic and Epigenetic Regulation of the Smoothened Gene (SMO) in Cancer Cells.Cancers (Basel)2020
31998314Tuning of NK-Specific HLA-C Expression by Alternative mRNA Splicing.Front Immunol2020
31231651The Human <i>TET2</i> Gene Contains Three Distinct Promoter Regions With Differing Tissue and Developmental Specificities.Front Cell Dev Biol2019
31471108Differential Activation of the Transcription Factor IRF1 Underlies the Distinct Immune Responses Elicited by Type I and Type III Interferons.Immunity2019
29329284Identification of an elaborate NK-specific system regulating HLA-C expression.PLoS Genet2018
30270560Identification of trophoblast-specific elements in the HLA-C core promoter.HLA2018
29559979Association of <i>TNFRSF1B</i> Promoter Polymorphisms with Human Disease: Further Studies Examining T-Regulatory Cells Are Required.Front Immunol2018
27819678TET2 binds the androgen receptor and loss is associated with prostate cancer.Oncogene2017
27467282Analysis of Ly49 gene transcripts in mature NK cells supports a role for the Pro1 element in gene activation, not gene expression.Genes Immun2016
26656451Characterization of KIR intermediate promoters reveals four promoter types associated with distinct expression patterns of KIR subtypes.Genes Immun2016
27817866HLA-C Level Is Regulated by a Polymorphic Oct1 Binding Site in the HLA-C Promoter Region.Am J Hum Genet2016
28033606Correction: Activating Receptor Signals Drive Receptor Diversity in Developing Natural Killer Cells.PLoS Biol2016
27500644Activating Receptor Signals Drive Receptor Diversity in Developing Natural Killer Cells.PLoS Biol2016
24842371Transcriptional regulation of Munc13-4 expression in cytotoxic lymphocytes is disrupted by an intronic mutation associated with a primary immunodeficiency.J Exp Med2014
25474109Interleukin-1 and interferon-γ orchestrate β-glucan-activated human dendritic cell programming via IκB-ζ modulation.PLoS One2014
24989671Characterization of a weakly expressed KIR2DL1 variant reveals a novel upstream promoter that controls KIR expression.Genes Immun2014
23675302LAB/NTAL facilitates fungal/PAMP-induced IL-12 and IFN-γ production by repressing β-catenin activation in dendritic cells.PLoS Pathog2013
23863987Identification of a KIR antisense lncRNA expressed by progenitor cells.Genes Immun2013
22570471Inhibition of nicotinamide phosphoribosyltransferase (NAMPT) activity by small molecule GMX1778 regulates reactive oxygen species (ROS)-mediated cytotoxicity in a p53- and nicotinic acid phosphoribosyltransferase1 (NAPRT1)-dependent manner.J Biol Chem2012
23552698Genomic copy number alterations in clear cell renal carcinoma: associations with case characteristics and mechanisms of VHL gene inactivation.Oncogenesis2012
22661295Promoter variants in the MSMB gene associated with prostate cancer regulate MSMB/NCOA4 fusion transcripts.Hum Genet2012
20033655Identification and analysis of novel transcripts and promoters in the human killer cell immunoglobulin-like receptor (KIR) genes.Methods Mol Biol2010
18987359The transcription factor c-Myc enhances KIR gene transcription through direct binding to an upstream distal promoter element.Blood2009
19383797Fine mapping and functional analysis of a common variant in MSMB on chromosome 10q11.2 associated with prostate cancer susceptibility.Proc Natl Acad Sci U S A2009
19008943Genetic control of variegated KIR gene expression: polymorphisms of the bi-directional KIR3DL1 promoter are associated with distinct frequencies of gene expression.PLoS Genet2008
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Collaborators

Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 25
University of Virginia School of Medicine
Co-authored papers 7
National Cancer Institute
Co-authored papers 6
Frederick National Laboratory for Cancer Research, National Cancer Institute
Co-authored papers 5
Washington University School of Medicine
Co-authored papers 4
Frederick National Laboratory for Cancer Research
Co-authored papers 4
National Cancer Institute
Co-authored papers 4
National Cancer Institute, National Institutes of Health
Co-authored papers 3
National Cancer Institute
Co-authored papers 2
National Institute of Allergy and Infectious Diseases
Co-authored papers 2
Massachusetts General Hospital/Harvard Medical School
Co-authored papers 2
National Cancer Institute, National Institutes of Health, Inc.
Co-authored papers 2
National Cancer Institute
Co-authored papers 2
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 2
National Cancer Institute
Co-authored papers 2
National Cancer Institute
Co-authored papers 1
Co-authored papers 1
Co-authored papers 1
National Cancer Institute, National Institutes of Health
Co-authored papers 1
National Cancer Institute-Frederick
Co-authored papers 1
National Cancer Institute
Co-authored papers 1
Co-authored papers 1
Brigham and Women's Hospital
Co-authored papers 1
National Cancer Institute
Co-authored papers 1
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 1
University of Oxford
Co-authored papers 1
VA Boston Healthcare System
Co-authored papers 1
The Kolling Institute, University of Sydney, and Royal North Shore Hospital
Co-authored papers 1
Co-authored papers 1
National Cancer Institute.
Co-authored papers 1