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Author Details

Maximilian Kauer
St. Anna Children's Cancer Research Institute (CCRI)
2002
46
27
PMIDPaper TitleJournal TitlePublished Year
36054432Human repair-related Schwann cells adopt functions of antigen-presenting cells in vitro.Glia2022
36054432Human repair-related Schwann cells adopt functions of antigen-presenting cells in vitro.Glia2022
33419969YAP/TAZ inhibition reduces metastatic potential of Ewing sarcoma cells.Oncogenesis2021
33712610Schwann cell plasticity regulates neuroblastic tumor cell differentiation via epidermal growth factor-like protein 8.Nat Commun2021
33419969YAP/TAZ inhibition reduces metastatic potential of Ewing sarcoma cells.Oncogenesis2021
33712610Schwann cell plasticity regulates neuroblastic tumor cell differentiation via epidermal growth factor-like protein 8.Nat Commun2021
30296338JAG2 signaling induces differentiation of CD14<sup>+</sup> monocytes into Langerhans cell histiocytosis-like cells.J Leukoc Biol2019
30296338JAG2 signaling induces differentiation of CD14<sup>+</sup> monocytes into Langerhans cell histiocytosis-like cells.J Leukoc Biol2019
30705095CD371 cell surface expression: a unique feature of <i>DUX4</i>-rearranged acute lymphoblastic leukemia.Haematologica2019
31406155Retraction Note: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.Cell Death Dis2019
31406155Retraction Note: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.Cell Death Dis2019
30705095CD371 cell surface expression: a unique feature of <i>DUX4</i>-rearranged acute lymphoblastic leukemia.Haematologica2019
30038364Correction: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.Cell Death Dis2018
30123424Identifying the druggable interactome of EWS-FLI1 reveals MCL-1 dependent differential sensitivities of Ewing sarcoma cells to apoptosis inducers.Oncotarget2018
30038364Correction: Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.Cell Death Dis2018
30123424Identifying the druggable interactome of EWS-FLI1 reveals MCL-1 dependent differential sensitivities of Ewing sarcoma cells to apoptosis inducers.Oncotarget2018
28030800The role of miR-17-92 in the miRegulatory landscape of Ewing sarcoma.Oncotarget2017
28671673EWS-FLI1 perturbs MRTFB/YAP-1/TEAD target gene regulation inhibiting cytoskeletal autoregulatory feedback in Ewing sarcoma.Oncogene2017
28482719MEF2C-dysregulated pediatric T-cell acute lymphoblastic leukemia is associated with CDKN1B deletions and a poor response to glucocorticoid therapy.Leuk Lymphoma2017
28924177Loss of MAPK-activated protein kinase 2 enables potent dendritic cell-driven anti-tumour T cell response.Sci Rep2017
27760381High-throughput RNAi screen in Ewing sarcoma cells identifies leucine rich repeats and WD repeat domain containing 1 (LRWD1) as a regulator of EWS-FLI1 driven cell viability.Gene2017
28030800The role of miR-17-92 in the miRegulatory landscape of Ewing sarcoma.Oncotarget2017
28160567EWS-FLI1 confers exquisite sensitivity to NAMPT inhibition in Ewing sarcoma cells.Oncotarget2017
28482719MEF2C-dysregulated pediatric T-cell acute lymphoblastic leukemia is associated with CDKN1B deletions and a poor response to glucocorticoid therapy.Leuk Lymphoma2017
28160567EWS-FLI1 confers exquisite sensitivity to NAMPT inhibition in Ewing sarcoma cells.Oncotarget2017
28671673EWS-FLI1 perturbs MRTFB/YAP-1/TEAD target gene regulation inhibiting cytoskeletal autoregulatory feedback in Ewing sarcoma.Oncogene2017
28924177Loss of MAPK-activated protein kinase 2 enables potent dendritic cell-driven anti-tumour T cell response.Sci Rep2017
27760381High-throughput RNAi screen in Ewing sarcoma cells identifies leucine rich repeats and WD repeat domain containing 1 (LRWD1) as a regulator of EWS-FLI1 driven cell viability.Gene2017
27282934EWS-FLI1 impairs aryl hydrocarbon receptor activation by blocking tryptophan breakdown via the kynurenine pathway.FEBS Lett2016
27282934EWS-FLI1 impairs aryl hydrocarbon receptor activation by blocking tryptophan breakdown via the kynurenine pathway.FEBS Lett2016
26657295Metronomic topotecan impedes tumor growth of MYCN-amplified neuroblastoma cells in vitro and in vivo by therapy induced senescence.Oncotarget2016
26327566Genetic alterations in glucocorticoid signaling pathway components are associated with adverse prognosis in children with relapsed ETV6/RUNX1-positive acute lymphoblastic leukemia.Leuk Lymphoma2016
27735950Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.Cell Death Dis2016
26657295Metronomic topotecan impedes tumor growth of MYCN-amplified neuroblastoma cells in vitro and in vivo by therapy induced senescence.Oncotarget2016
26327566Genetic alterations in glucocorticoid signaling pathway components are associated with adverse prognosis in children with relapsed ETV6/RUNX1-positive acute lymphoblastic leukemia.Leuk Lymphoma2016
27735950Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.Cell Death Dis2016
25515960The role of the Janus-faced transcription factor PAX5-JAK2 in acute lymphoblastic leukemia.Blood2015
25712098EWS-FLI1 employs an E2F switch to drive target gene expression.Nucleic Acids Res2015
25410853Identification of RISC-associated adenoviral microRNAs, a subset of their direct targets, and global changes in the targetome upon lytic adenovirus 5 infection.J Virol2015
25515960The role of the Janus-faced transcription factor PAX5-JAK2 in acute lymphoblastic leukemia.Blood2015
26360775Enriched Bone Marrow Derived Disseminated Neuroblastoma Cells Can Be a Reliable Source for Gene Expression Studies-A Validation Study.PLoS One2015
26360775Enriched Bone Marrow Derived Disseminated Neuroblastoma Cells Can Be a Reliable Source for Gene Expression Studies-A Validation Study.PLoS One2015
25712098EWS-FLI1 employs an E2F switch to drive target gene expression.Nucleic Acids Res2015
25410853Identification of RISC-associated adenoviral microRNAs, a subset of their direct targets, and global changes in the targetome upon lytic adenovirus 5 infection.J Virol2015
23995784Suppression of FOXO1 is responsible for a growth regulatory repressive transcriptional sub-signature of EWS-FLI1 in Ewing sarcoma.Oncogene2014
25281719Suppression of deacetylase SIRT1 mediates tumor-suppressive NOTCH response and offers a novel treatment option in metastatic Ewing sarcoma.Cancer Res2014
23995784Suppression of FOXO1 is responsible for a growth regulatory repressive transcriptional sub-signature of EWS-FLI1 in Ewing sarcoma.Oncogene2014
24240203Blocking ETV6/RUNX1-induced MDM2 overexpression by Nutlin-3 reactivates p53 signaling in childhood leukemia.Leukemia2014
25281719Suppression of deacetylase SIRT1 mediates tumor-suppressive NOTCH response and offers a novel treatment option in metastatic Ewing sarcoma.Cancer Res2014
24240203Blocking ETV6/RUNX1-induced MDM2 overexpression by Nutlin-3 reactivates p53 signaling in childhood leukemia.Leukemia2014
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Collaborators

St. Anna Children's Cancer Research Institute (CCRI), Medical University Vienna
Co-authored papers 23
Children's Cancer Research Institute
Co-authored papers 16
Children's Cancer Research Institute
Co-authored papers 10
Medical University of Innsbruck
Co-authored papers 7
Children's Cancer Research Institute
Co-authored papers 6
St. Anna Children's Cancer Research Institute (CCRI)
Co-authored papers 5
Georgetown University Medical Center
Co-authored papers 4
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 4
Children's Cancer Research Institute, Medical University of Vienna
Co-authored papers 4
St. Anna Children's Cancer Research Institute
Co-authored papers 4
Children's Cancer Research Center
Co-authored papers 3
Institute of Animal Breeding and Genetics, University of Veterinary Medicine Vienna
Co-authored papers 3
St. Anna Children's Cancer Research Institute
Co-authored papers 3
St. Anna Children's Cancer Research Institute (CCRI)
Co-authored papers 3
CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences
Co-authored papers 3
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 2
West China Second Hospital, Sichuan University
Co-authored papers 2
Children's Cancer Research Institute, Medical University of Vienna
Co-authored papers 2
Johns Hopkins School of Medicine, National Cancer Institute, Princeton University, University of Colorado Anschutz Medical Campus, University of Pittsburgh, University of Washington
Co-authored papers 2
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VTT Technical Research Centre of Finland Ltd
Co-authored papers 2
Seattle Children's Research Institute
Co-authored papers 2
Medical University of Vienna
Co-authored papers 2
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Co-authored papers 2
IRCCS Istituto Ortopedico Rizzoli
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St. Anna Children's Cancer Research Institute (CCRI), Medical University of Vienna
Co-authored papers 2
University College London, Cancer Institute
Co-authored papers 1
Georgetown University Medical Center, Georgetown University
Co-authored papers 1
National Cancer Institute, National Institutes of Health
Co-authored papers 1
Cell Therapy Institute, Paracelsus Medical University (PMU)
Co-authored papers 1