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Author Details
Full Name
Matthew D Storck
Affiliation
Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska
ORCID
Career Start Year
2015
Papers
25
H Index
10
Expertise
CM4AI Collaborator
Nevan J Krogan (CM4AI)
PMID
Paper Title
Journal Title
Published Year
36993422
GD2 and its biosynthetic enzyme GD3 synthase promote tumorigenesis in prostate cancer by regulating cancer stem cell behavior.
bioRxiv
2024
36711452
EHD1-dependent traffic of IGF-1 receptor to the cell surface is essential for Ewing sarcoma tumorigenesis and metastasis.
bioRxiv
2023
37292716
Cbl and Cbl-b Ubiquitin Ligases are Essential for Intestinal Epithelial Stem Cell Maintenance.
bioRxiv
2023
37474760
EHD1-dependent traffic of IGF-1 receptor to the cell surface is essential for Ewing sarcoma tumorigenesis and metastasis.
Commun Biol
2023
36625722
EHD2 overexpression promotes tumorigenesis and metastasis in triple-negative breast cancer by regulating store-operated calcium entry.
Elife
2023
35675041
Ecdysoneless Overexpression Drives Mammary Tumorigenesis through Upregulation of C-MYC and Glucose Metabolism.
Mol Cancer Res
2022
34439093
CHIP/STUB1 Ubiquitin Ligase Functions as a Negative Regulator of ErbB2 by Promoting Its Early Post-Biosynthesis Degradation.
Cancers (Basel)
2021
31932478
EHD1 and RUSC2 Control Basal Epidermal Growth Factor Receptor Cell Surface Expression and Recycling.
Mol Cell Biol
2020
29212907
Role of the EHD Family of Endocytic Recycling Regulators for TCR Recycling and T Cell Function.
J Immunol
2018
30118682
An Mtb-Human Protein-Protein Interaction Map Identifies a Switch between Host Antiviral and Antibacterial Responses.
Mol Cell
2018
30389896
Correction: An essential role of CBL and CBL-B ubiquitin ligases in mammary stem cell maintenance (doi: 10.1242/dev.138164).
Development
2018
29510992
Loss of the Nuclear Pool of Ubiquitin Ligase CHIP/STUB1 in Breast Cancer Unleashes the MZF1-Cathepsin Pro-oncogenic Program.
Cancer Res
2018
28778975
EHD4 is a novel regulator of urinary water homeostasis.
FASEB J
2017
28082680
Structural Determinants of the Gain-of-Function Phenotype of Human Leukemia-associated Mutant CBL Oncogene.
J Biol Chem
2017
28332491
Corrigendum: Endocytic recycling protein EHD1 regulates primary cilia morphogenesis and SHH signaling during neural tube development.
Sci Rep
2017
28100467
An essential role of CBL and CBL-B ubiquitin ligases in mammary stem cell maintenance.
Development
2017
26177294
Fasudil, a clinically safe ROCK inhibitor, decreases disease burden in a Cbl/Cbl-b deficiency-driven murine model of myeloproliferative disorders.
Hematology
2016
27449297
VAV1-Cre mediated hematopoietic deletion of CBL and CBL-B leads to JMML-like aggressive early-neonatal myeloproliferative disease.
Oncotarget
2016
27276677
A novel CBL-Bflox/flox mouse model allows tissue-selective fully conditional CBL/CBL-B double-knockout: CD4-Cre mediated CBL/CBL-B deletion occurs in both T-cells and hematopoietic stem cells.
Oncotarget
2016
27510742
Corrigendum to CSF-1 receptor signalling is governed by pre-requisite EHD1 mediated receptor display on the macrophage cell surface [Cell Signalling 2016 Sep.; 28(9): 1325-35].
Cell Signal
2016
26859680
Marked enhancement of lysosomal targeting and efficacy of ErbB2-targeted drug delivery by HSP90 inhibition.
Oncotarget
2016
26884322
Endocytic recycling protein EHD1 regulates primary cilia morphogenesis and SHH signaling during neural tube development.
Sci Rep
2016
25660204
Polypeptide-based nanogels co-encapsulating a synergistic combination of doxorubicin with 17-AAG show potent anti-tumor activity in ErbB2-driven breast cancer models.
J Control Release
2015
25871390
Loss of Cbl and Cbl-b ubiquitin ligases abrogates hematopoietic stem cell quiescence and sensitizes leukemic disease to chemotherapy.
Oncotarget
2015
26455409
The endocytic recycling regulatory protein EHD1 Is required for ocular lens development.
Dev Biol
2015
1 - 25 of 25
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1
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University of California san francisco
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University of California berkeley
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Gladstone Institutes
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School of Public Health, University of California berkeley
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University of California san francisco
Co-authored papers
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University of Wyoming
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