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Author Details

Cherilyn A Elwell
University of California san francisco
2000
17
14
Nevan J Krogan (CM4AI)
PMIDPaper TitleJournal TitlePublished Year
29703496Emerging Role of Retromer in Modulating Pathogen Growth.Trends Microbiol2018
30314703Both the N- and C- terminal regions of the Chlamydial inclusion protein D (IncD) are required for interaction with the pleckstrin homology domain of the ceramide transport protein CERT.Biochem Biophys Res Commun2018
28252385<i>Chlamydia</i> interfere with an interaction between the mannose-6-phosphate receptor and sorting nexins to counteract host restriction.Elife2017
27108705Chlamydia cell biology and pathogenesis.Nat Rev Microbiol2016
26118995Global Mapping of the Inc-Human Interactome Reveals that Retromer Restricts Chlamydia Infection.Cell Host Microbe2015
23480519Endosulfatases SULF1 and SULF2 limit Chlamydia muridarum infection.Cell Microbiol2013
23637308Chlamydial intracellular survival strategies.Cold Spring Harb Perspect Med2013
22452394Lipid acquisition by intracellular Chlamydiae.Cell Microbiol2012
21586644Species-specific interactions of Src family tyrosine kinases regulate Chlamydia intracellular growth and trafficking.mBio2011
21998584Chlamydia trachomatis co-opts the FGF2 signaling pathway to enhance infection.PLoS Pathog2011
21909260Chlamydia trachomatis co-opts GBF1 and CERT to acquire host sphingomyelin for distinct roles during intracellular development.PLoS Pathog2011
18369471RNA interference screen identifies Abl kinase and PDGFR signaling in Chlamydia trachomatis entry.PLoS Pathog2008
15839901Drosophila melanogaster S2 cells: a model system to study Chlamydia interaction with host cells.Cell Microbiol2005
14617801RNA interference inhibition of Mus81 reduces mitotic recombination in human cells.Mol Biol Cell2004
15557670Lipid raft-mediated entry is not required for Chlamydia trachomatis infection of cultured epithelial cells.Infect Immun2004
11292766Escherichia coli CdtB mediates cytolethal distending toxin cell cycle arrest.Infect Immun2001
10972814DNase I homologous residues in CdtB are critical for cytolethal distending toxin-mediated cell cycle arrest.Mol Microbiol2000
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Collaborators

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Genentech Inc.
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Duke University
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Gladstone Institutes
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College of Osteopathic Medicine, Michigan State University
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University of California san francisco
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Complete Genomics Inc.
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Duke University
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University of California Davis
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Seattle Children's Research Institute
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University of California san francisco
Co-authored papers 1
Quantitative Biosciences Institute (QBI), University of California san francisco
Co-authored papers 1
University of California berkeley
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The Scripps Research Institute
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