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Author Details
Full Name
Benjamin E Nilsson-Payant
Affiliation
Hanover Medical School
ORCID
Career Start Year
2017
Papers
27
H Index
16
Expertise
CM4AI Collaborator
Kirsten Obernier (CM4AI)
PMID
Paper Title
Journal Title
Published Year
36918693
A multi-organoid platform identifies CIART as a key factor for SARS-CoV-2 infection.
Nat Cell Biol
2023
37876781
Modulation of Influenza A virus NS1 expression reveals prioritization of host response antagonism at single-cell resolution.
Front Microbiol
2023
37345956
SARS-CoV-2 hijacks p38β/MAPK11 to promote virus replication.
mBio
2023
35294338
Coagulation factors directly cleave SARS-CoV-2 spike and enhance viral entry.
Elife
2022
36570071
Dual-Reporter System for Real-Time Monitoring of SARS-CoV-2 Main Protease Activity in Live Cells Enables Identification of an Allosteric Inhibition Path.
ACS Bio Med Chem Au
2022
36206731
A human iPSC-array-based GWAS identifies a virus susceptibility locus in the NDUFA4 gene and functional variants.
Cell Stem Cell
2022
36282911
Host protein kinases required for SARS-CoV-2 nucleocapsid phosphorylation and viral replication.
Sci Signal
2022
36083891
Sensing of SARS-CoV-2 by pDCs and their subsequent production of IFN-I contribute to macrophage-induced cytokine storm during COVID-19.
Sci Immunol
2022
35255712
SARS-CoV-2 Infection Induces Ferroptosis of Sinoatrial Node Pacemaker Cells.
Circ Res
2022
34935435
The Host Factor ANP32A Is Required for Influenza A Virus vRNA and cRNA Synthesis.
J Virol
2022
33116299
Identification of SARS-CoV-2 inhibitors using lung and colonic organoids.
Nature
2021
33853355
An Immuno-Cardiac Model for Macrophage-Mediated Inflammation in COVID-19 Hearts.
Circ Res
2021
33568513
Reduced Nucleoprotein Availability Impairs Negative-Sense RNA Virus Replication and Promotes Host Recognition.
J Virol
2021
34644531
Cardiomyocytes recruit monocytes upon SARS-CoV-2 infection by secreting CCL2.
Stem Cell Reports
2021
34523966
The NF-κB Transcriptional Footprint Is Essential for SARS-CoV-2 Replication.
J Virol
2021
34403650
Cardiomyocytes recruit monocytes upon SARS-CoV-2 infection by secreting CCL2.
Stem Cell Reports
2021
33941899
A human-airway-on-a-chip for the rapid identification of candidate antiviral therapeutics and prophylactics.
Nat Biomed Eng
2021
34180223
Zinc-Embedded Polyamide Fabrics Inactivate SARS-CoV-2 and Influenza A Virus.
ACS Appl Mater Interfaces
2021
32511403
Identification of Candidate COVID-19 Therapeutics using hPSC-derived Lung Organoids.
bioRxiv
2020
33236003
SARS-CoV-2 Infected Cardiomyocytes Recruit Monocytes by Secreting CCL2.
Res Sq
2020
32579880
A Human Pluripotent Stem Cell-based Platform to Study SARS-CoV-2 Tropism and Model Virus Infection in Human Cells and Organoids.
Cell Stem Cell
2020
32645325
The Global Phosphorylation Landscape of SARS-CoV-2 Infection.
Cell
2020
32416070
Imbalanced Host Response to SARS-CoV-2 Drives Development of COVID-19.
Cell
2020
29338047
Amino acid substitutions affecting aspartic acid 605 and valine 606 decrease the interaction strength between the influenza virus RNA polymerase PB2 '627' domain and the viral nucleoprotein.
PLoS One
2018
29875249
The Surface-Exposed PA<sup>51-72</sup>-Loop of the Influenza A Virus Polymerase Is Required for Viral Genome Replication.
J Virol
2018
28550113
Erratum for Nilsson et al., "Role of the PB2 627 Domain in Influenza A Virus Polymerase Function".
J Virol
2017
28122973
Role of the PB2 627 Domain in Influenza A Virus Polymerase Function.
J Virol
2017
1 - 27 of 27
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