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Author Details

Mara Rosenberg
Broad Institute of MIT and Harvard
2013
44
29
PMIDPaper TitleJournal TitlePublished Year
35084050Implementation of a prostate cancer-specific targeted sequencing panel for credentialing of patient-derived cell lines and genomic characterization of patient samples.Prostate2022
36307214Comprehensive molecular characterization of a rare case of Philadelphia chromosome-positive acute myeloid leukemia.Cold Spring Harb Mol Case Stud2022
35084050Implementation of a prostate cancer-specific targeted sequencing panel for credentialing of patient-derived cell lines and genomic characterization of patient samples.Prostate2022
36307214Comprehensive molecular characterization of a rare case of Philadelphia chromosome-positive acute myeloid leukemia.Cold Spring Harb Mol Case Stud2022
32302173Constraints on Lorentz Invariance Violation from HAWC Observations of Gamma Rays above 100 TeV.Phys Rev Lett2020
32004015Multiple Galactic Sources with Emission Above 56 TeV Detected by HAWC.Phys Rev Lett2020
32302173Constraints on Lorentz Invariance Violation from HAWC Observations of Gamma Rays above 100 TeV.Phys Rev Lett2020
32754299Genomic markers of midostaurin drug sensitivity in FLT3 mutated and FLT3 wild-type acute myeloid leukemia patients.Oncotarget2020
33028839Framework for quality assessment of whole genome cancer sequences.Nat Commun2020
32004015Multiple Galactic Sources with Emission Above 56 TeV Detected by HAWC.Phys Rev Lett2020
33028839Framework for quality assessment of whole genome cancer sequences.Nat Commun2020
32754299Genomic markers of midostaurin drug sensitivity in FLT3 mutated and FLT3 wild-type acute myeloid leukemia patients.Oncotarget2020
30425048CSF1R inhibitors exhibit antitumor activity in acute myeloid leukemia by blocking paracrine signals from support cells.Blood2019
30425048CSF1R inhibitors exhibit antitumor activity in acute myeloid leukemia by blocking paracrine signals from support cells.Blood2019
31164371Genomic Evolutionary Patterns of Leiomyosarcoma and Liposarcoma.Clin Cancer Res2019
31048320The TP53 Apoptotic Network Is a Primary Mediator of Resistance to BCL2 Inhibition in AML Cells.Cancer Discov2019
31145495The combination of NPM1, DNMT3A, and IDH1/2 mutations leads to inferior overall survival in AML.Am J Hematol2019
31145495The combination of NPM1, DNMT3A, and IDH1/2 mutations leads to inferior overall survival in AML.Am J Hematol2019
31164371Genomic Evolutionary Patterns of Leiomyosarcoma and Liposarcoma.Clin Cancer Res2019
31048320The TP53 Apoptotic Network Is a Primary Mediator of Resistance to BCL2 Inhibition in AML Cells.Cancer Discov2019
29713087Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes.Nat Med2018
30482938Publisher Correction: Very-high-energy particle acceleration powered by the jets of the microquasar SS 433.Nature2018
29955181Publisher Correction: Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes.Nat Med2018
29955182Author Correction: Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes.Nat Med2018
30283106Very-high-energy particle acceleration powered by the jets of the microquasar SS 433.Nature2018
29941871DeTiN: overcoming tumor-in-normal contamination.Nat Methods2018
29713087Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes.Nat Med2018
29941871DeTiN: overcoming tumor-in-normal contamination.Nat Methods2018
29955181Publisher Correction: Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes.Nat Med2018
29955182Author Correction: Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomes.Nat Med2018
30482938Publisher Correction: Very-high-energy particle acceleration powered by the jets of the microquasar SS 433.Nature2018
30283106Very-high-energy particle acceleration powered by the jets of the microquasar SS 433.Nature2018
28522256Sporadic Early-Onset Diffuse Gastric Cancers Have High Frequency of Somatic CDH1 Alterations, but Low Frequency of Somatic RHOA Mutations Compared With Late-Onset Cancers.Gastroenterology2017
28658208Recurrent and functional regulatory mutations in breast cancer.Nature2017
28522256Sporadic Early-Onset Diffuse Gastric Cancers Have High Frequency of Somatic CDH1 Alterations, but Low Frequency of Somatic RHOA Mutations Compared With Late-Onset Cancers.Gastroenterology2017
29109393Scalable whole-exome sequencing of cell-free DNA reveals high concordance with metastatic tumors.Nat Commun2017
29146808Extended gamma-ray sources around pulsars constrain the origin of the positron flux at Earth.Science2017
29109393Scalable whole-exome sequencing of cell-free DNA reveals high concordance with metastatic tumors.Nat Commun2017
29146808Extended gamma-ray sources around pulsars constrain the origin of the positron flux at Earth.Science2017
28658208Recurrent and functional regulatory mutations in breast cancer.Nature2017
26711114Corrigendum: The genomic landscape of juvenile myelomonocytic leukemia.Nat Genet2016
27348297The genomic landscape and evolution of endometrial carcinoma progression and abdominopelvic metastasis.Nat Genet2016
27149842Genomic Correlates of Immune-Cell Infiltrates in Colorectal Carcinoma.Cell Rep2016
27167109A comparative assessment of clinical whole exome and transcriptome profiling across sequencing centers: implications for precision cancer medicine.Oncotarget2016
27158780Distinct patterns of somatic genome alterations in lung adenocarcinomas and squamous cell carcinomas.Nat Genet2016
26711114Corrigendum: The genomic landscape of juvenile myelomonocytic leukemia.Nat Genet2016
27760322Genomic Correlates of Immune-Cell Infiltrates in Colorectal Carcinoma.Cell Rep2016
27760322Genomic Correlates of Immune-Cell Infiltrates in Colorectal Carcinoma.Cell Rep2016
27149842Genomic Correlates of Immune-Cell Infiltrates in Colorectal Carcinoma.Cell Rep2016
27167109A comparative assessment of clinical whole exome and transcriptome profiling across sequencing centers: implications for precision cancer medicine.Oncotarget2016
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Collaborators

Broad Institute of the Massachusetts Institute of Technology and Harvard
Co-authored papers 28
Broad Institute of MIT and Harvard
Co-authored papers 17
Co-authored papers 16
Co-authored papers 16
Broad Institute
Co-authored papers 14
Co-authored papers 13
Dana-Farber Cancer Institute
Co-authored papers 12
Broad Institute of Harvard and MIT
Co-authored papers 11
Broad Institute of MIT and Harvard
Co-authored papers 10
Broad Institute of MIT and Harvard
Co-authored papers 10
Co-authored papers 10
Co-authored papers 9
Co-authored papers 8
Vanderbilt-Ingram Cancer Center, Vanderbilt University
Co-authored papers 7
Co-authored papers 7
Co-authored papers 7
Broad Institute
Co-authored papers 6
Co-authored papers 6
Broad Institute of MIT and Harvard
Co-authored papers 5
Broad Institute of Massachusetts Institute of Technology and Harvard
Co-authored papers 5
The Broad Institute of Harvard and MIT
Co-authored papers 5
Dana-Farber Cancer Institute
Co-authored papers 5
Co-authored papers 5
University of Maryland, College Park
Co-authored papers 5
Co-authored papers 5
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The Broad Institute of MIT and Harvard
Co-authored papers 5
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