Skip to Main Content

Author Details

Anderly C Chueh
Biomedicine Discovery Institute, Monash University
2005
28
13
PMIDPaper TitleJournal TitlePublished Year
36765160Author Correction: DUSP5 is methylated in CIMP-high colorectal cancer but is not a major regulator of intestinal cell proliferation and tumorigenesis.Sci Rep2023
37712140Correction: ATF3 Repression of BCL-XL Determines Apoptotic Sensitivity to HDAC Inhibitors Across Tumor Types.Clin Cancer Res2023
37557181Discovery of a highly potent, selective, orally bioavailable inhibitor of KAT6A/B histone acetyltransferases with efficacy against KAT6A-high ER+ breast cancer.Cell Chem Biol2023
38066546Pediatric glioma histone H3.3 K27M/G34R mutations drive abnormalities in PML nuclear bodies.Genome Biol2023
36608258Integrative characterisation of secreted factors involved in intercellular communication between prostate epithelial or cancer cells and fibroblasts.Mol Oncol2023
35075138VEGF-A, VEGFR1 and VEGFR2 single nucleotide polymorphisms and outcomes from the AGITG MAX trial of capecitabine, bevacizumab and mitomycin C in metastatic colorectal cancer.Sci Rep2022
36550548Identification of biological pathways and processes regulated by NEK5 in breast epithelial cells via an integrated proteomic approach.Cell Commun Signal2022
33712556Preclinical small molecule WEHI-7326 overcomes drug resistance and elicits response in patient-derived xenograft models of human treatment-refractory tumors.Cell Death Dis2021
33547865Proteomics-based interrogation of the kinome and its implications for precision oncology.Proteomics2021
33767439CSK-homologous kinase (CHK/MATK) is a potential colorectal cancer tumour suppressor gene epigenetically silenced by promoter methylation.Oncogene2021
29379130DUSP5 is methylated in CIMP-high colorectal cancer but is not a major regulator of intestinal cell proliferation and tumorigenesis.Sci Rep2018
28611196ATF3 Repression of BCL-X<sub>L</sub> Determines Apoptotic Sensitivity to HDAC Inhibitors across Tumor Types.Clin Cancer Res2017
29088766Promoter hypomethylation of NY-ESO-1, association with clinicopathological features and PD-L1 expression in non-small cell lung cancer.Oncotarget2017
28784162Csk-homologous kinase (Chk) is an efficient inhibitor of Src-family kinases but a poor catalyst of phosphorylation of their C-terminal regulatory tyrosine.Cell Commun Signal2017
26899498A beacon of hope in stroke therapy-Blockade of pathologically activated cellular events in excitotoxic neuronal death as potential neuroprotective strategies.Pharmacol Ther2016
26983878Dual Targeting of Bromodomain and Extraterminal Domain Proteins, and WNT or MAPK Signaling, Inhibits c-MYC Expression and Proliferation of Colorectal Cancer Cells.Mol Cancer Ther2016
26907172Transketolase-like 1 ectopic expression is associated with DNA hypomethylation and induces the Warburg effect in melanoma cells.BMC Cancer2016
24632621PR55α-containing protein phosphatase 2A complexes promote cancer cell migration and invasion through regulation of AP-1 transcriptional activity.Oncogene2015
26092983The intestinal epithelial cell differentiation marker intestinal alkaline phosphatase (ALPi) is selectively induced by histone deacetylase inhibitors (HDACi) in colon cancer cells in a Kruppel-like factor 5 (KLF5)-dependent manner.J Biol Chem2015
25740609PR55α-containing protein phosphatase 2A complexes promote cancer cell migration and invasion through regulation of AP-1 transcriptional activity.Oncogene2015
24512308Mechanisms of Histone Deacetylase Inhibitor-Regulated Gene Expression in Cancer Cells.Antioxid Redox Signal2015
24406338FOXP3 over-expression inhibits melanoma tumorigenesis via effects on proliferation and apoptosis.Oncotarget2014
24045184Molecular imaging of death receptor 5 occupancy and saturation kinetics in vivo by humanized monoclonal antibody CS-1008.Clin Cancer Res2013
20068171Apoptotic sensitivity of colon cancer cells to histone deacetylase inhibitors is mediated by an Sp1/Sp3-activated transcriptional program involving immediate-early gene induction.Cancer Res2010
19180186LINE retrotransposon RNA is an essential structural and functional epigenetic component of a core neocentromeric chromatin.PLoS Genet2009
18252209Neocentromeres: new insights into centromere structure, disease development, and karyotype evolution.Am J Hum Genet2008
15537667Variable and hierarchical size distribution of L1-retroelement-enriched CENP-A clusters within a functional human neocentromere.Hum Mol Genet2005
15832308BAC-based PCR fragment microarray: high-resolution detection of chromosomal deletion and duplication breakpoints.Hum Mutat2005
  • 1 - 28 of 28

Recommended Authors

Ottawa Hospital Research Institute
Career Start Year 2010
Number of shared co-authors 1
College of Medicine, University of Saskatchewan
Career Start Year 2010
Number of shared co-authors 1
McDermott Center for Human Growth and Development.
Career Start Year 2010
Number of shared co-authors 0
Princess Margaret Cancer Centre, University Health Network
Career Start Year 2010
Number of shared co-authors 1
Montefiore Medical Center
Career Start Year 2008
Number of shared co-authors 0
The University of Texas MD Anderson Cancer Center
Career Start Year 2006
Number of shared co-authors 0
Memorial Sloan Kettering Cancer Center
Career Start Year 2005
Number of shared co-authors 0
Cancer Science Institute of Singapore, National University of Singapore
Career Start Year 2005
Number of shared co-authors 0
The University of Texas MD Anderson Cancer Center
Career Start Year 2005
Number of shared co-authors 0
University of Nebraska Medical Center
Career Start Year 2005
Number of shared co-authors 0
National Center for Advancing Translational Sciences
Career Start Year 2004
Number of shared co-authors 0
Collegeville
Career Start Year 2003
Number of shared co-authors 0
QIMR Berghofer Medical Research Institute
Career Start Year 2003
Number of shared co-authors 1
Roswell Park Comprehensive Cancer Center
Career Start Year 2003
Number of shared co-authors 0
The Wistar Institute
Career Start Year 2003
Number of shared co-authors 0
National Center for Advancing Translational Sciences
Career Start Year 2003
Number of shared co-authors 0
Fox Chase Cancer Center
Career Start Year 2002
Number of shared co-authors 0
University of Alabama at Birmingham, USA O'Neal Comprehensive Cancer Center
Career Start Year 2001
Number of shared co-authors 0
Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai
Career Start Year 2001
Number of shared co-authors 0
Stanford Center for Biomedical Informatics Research, Stanford University
Career Start Year 1999
Number of shared co-authors 0
Roswell Park Comprehensive Cancer Center
Career Start Year 1999
Number of shared co-authors 0
Institute for Translational Epigenetics, University of Cologne
Career Start Year 1999
Number of shared co-authors 0
Center for Cancer Research, National Cancer Institute, National Institutes of Health
Career Start Year 1997
Number of shared co-authors 0
The University of Texas MD Anderson Cancer Center
Career Start Year 1997
Number of shared co-authors 1
Cleveland Clinic.
Career Start Year 1994
Number of shared co-authors 2
The University of Melbourne
Career Start Year 1990
Number of shared co-authors 2
Broad Institute of MIT and Harvard
Career Start Year 1989
Number of shared co-authors 0
Broad Institute of MIT and Harvard, USA Dana-Farber Cancer Institute
Career Start Year 1987
Number of shared co-authors 0
Broad Institute
Career Start Year 1983
Number of shared co-authors 2
German Cancer Research Center
Career Start Year 1982
Number of shared co-authors 1

Collaborators

Olivia Newton-John Cancer Research Institute
Co-authored papers 15
The University of Queensland
Co-authored papers 4
Co-authored papers 2
Olivia Newton-John Cancer Research Institute
Co-authored papers 2
Australia Olivia Newton-John Cancer Research Institute, La Trobe University
Co-authored papers 2
Olivia Newton-John Cancer Research Institute
Co-authored papers 2
St Vincent's Hospital
Co-authored papers 1
National Institutes of Health
Co-authored papers 1
Co-authored papers 1
Co-authored papers 1
Co-authored papers 1
Ontario Institute for Cancer Research
Co-authored papers 1
Co-authored papers 1
Royal Melbourne Hospital
Co-authored papers 1
Olivia Newton-John Cancer Research Institute
Co-authored papers 1
University of California los angeles
Co-authored papers 1
St. Vincent's Hospital, University of Melbourne
Co-authored papers 1
Co-authored papers 1
Co-authored papers 1
Walter and Eliza Hall Institute of Medical Research
Co-authored papers 1
The University of Melbourne
Co-authored papers 1
Peter MacCallum Cancer Centre
Co-authored papers 1
Peter MacCallum Cancer Centre
Co-authored papers 1
Co-authored papers 1
School of Clinical Medicine
Co-authored papers 1