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Author Details

Shaojian Gao
1996
24
16
PMIDPaper TitleJournal TitlePublished Year
34638461Alterations in HLA Class I-Presented Immunopeptidome and Class I-Interactome upon Osimertinib Resistance in EGFR Mutant Lung Adenocarcinoma.Cancers2021
33727228Alterations in the Global Proteome and Phosphoproteome in Third Generation EGFR TKI Resistance Reveal Drug Targets to Circumvent Resistance.Cancer Res2021
34391887Proteogenomic Analysis Unveils the HLA Class I-Presented Immunopeptidome in Melanoma and EGFR-Mutant Lung Adenocarcinoma.Mol Cell Proteomics2021
32483558Clonal Evolution and Heterogeneity of Osimertinib Acquired Resistance Mechanisms in EGFR Mutant Lung Cancer.Cell Rep Med2020
31783178Anaplastic Lymphoma Kinase Gene Rearrangement in Children and Young Adults With Mesothelioma.J Thorac Oncol2020
31570787Bile acid-induced "Minority MOMP" promotes esophageal carcinogenesis while maintaining apoptotic resistance via Mcl-1.Oncogene2020
30840888APOBEC Mutagenesis and Copy-Number Alterations Are Drivers of Proteogenomic Tumor Evolution and Heterogeneity in Metastatic Thoracic Tumors.Cell Rep2019
31740700Integration of TGF-β-induced Smad signaling in the insulin-induced transcriptional response in endothelial cells.Scientific Reports2019
30617155Quantitative Mass Spectrometry to Interrogate Proteomic Heterogeneity in Metastatic Lung Adenocarcinoma and Validate a Novel Somatic Mutation CDK12-G879V.Mol Cell Proteomics2019
30975761Inherited predisposition to malignant mesothelioma and overall survival following platinum chemotherapy.Proc Natl Acad Sci U S A2019
30455306Discovery of Kaposi's sarcoma herpesvirus-encoded circular RNAs and a human antiviral circular RNA.Proceedings of the National Academy of Sciences of the United States of America2018
29934340Induction of Thioredoxin-Interacting Protein by a Histone Deacetylase Inhibitor, Entinostat, Is Associated with DNA Damage and Apoptosis in Esophageal Adenocarcinoma.Mol Cancer Ther2018
29036671CRISPR/Cas9-mediated gene knockout is insensitive to target copy number but is dependent on guide RNA potency and Cas9/sgRNA threshold expression level.Nucleic Acids Res2017
28334828Flexible CRISPR library construction using parallel oligonucleotide retrieval.Nucleic Acids Research2017
28331001Quantitative Tyrosine Phosphoproteomics of Epidermal Growth Factor Receptor (EGFR) Tyrosine Kinase Inhibitor-treated Lung Adenocarcinoma Cells Reveals Potential Novel Biomarkers of Therapeutic Response.Mol Cell Proteomics2017
27776121Wild-Type U2AF1 Antagonizes the Splicing Program Characteristic of U2AF1-Mutant Tumors and Is Required for Cell Survival.PLoS Genet2016
27900369Genomic profiling of multiple sequentially acquired tumor metastatic sites from an "exceptional responder" lung adenocarcinoma patient reveals extensive genomic heterogeneity and novel somatic variants driving treatment response.Cold Spring Harb Mol Case Stud2016
27746021Direct Regulation of Alternative Splicing by SMAD3 through PCBP1 Is Essential to the Tumor-Promoting Role of TGF-β.Molecular Cell2016
27912093Direct Regulation of Alternative Splicing by SMAD3 through PCBP1 Is Essential to the Tumor-Promoting Role of TGF-β.Molecular Cell2016
25030918Using pooled miR30-shRNA library for cancer lethal and synthetic lethal screens.Methods in Molecular Biology2014
9805400Mutagenesis of putative acylation sites alters function, localization, and accumulation of a Gi alpha subunit of the chestnut blight fungus Cryphonectria parasitica.Molecular Plant-Microbe Interactions1998
8755591Extensive alteration of fungal gene transcript accumulation and elevation of G-protein-regulated cAMP levels by a virulence-attenuating hypovirus.Proceedings of the National Academy of Sciences of the United States of America1996
8787397Cloning and targeted disruption of enpg-1, encoding the major in vitro extracellular endopolygalacturonase of the chestnut blight fungus, Cryphonectria parasitica.Applied and Environmental Microbiology1996
11038529Distinct roles for two G protein alpha subunits in fungal virulence, morphology, and reproduction revealed by targeted gene disruption.Proceedings of the National Academy of Sciences of the United States of America1996
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