|
| 36114991 | PU.1 inhibition does not attenuate cardiac function deterioration or fibrosis in a murine model of myocardial infarction. | Molecular and Cellular Biochemistry | 2023 |
| 35061545 | Transient Cell Cycle Induction in Cardiomyocytes to Treat Subacute Ischemic Heart Failure. | Circulation | 2022 |
| 34948245 | Effects of Heme Oxygenase-1 on c-Kit-Positive Cardiac Cells. | International Journal of Molecular Sciences | 2021 |
| 33808720 | Comparison of Repeated Doses of C-kit-Positive Cardiac Cells versus a Single Equivalent Combined Dose in a Murine Model of Chronic Ischemic Cardiomyopathy. | International Journal of Molecular Sciences | 2021 |
| 34287784 | Single dose of synthetic microRNA-199a or microRNA-149 mimic does not improve cardiac function in a murine model of myocardial infarction. | Molecular and Cellular Biochemistry | 2021 |
| 34124043 | Cardiac Mesenchymal Cells Cultured at Physiologic Oxygen Tension Have Superior Therapeutic Efficacy in Heart Failure Caused by Myocardial Infarction. | Frontiers in Cell and Developmental Biology | 2021 |
| 33547546 | Echocardiography-guided percutaneous left ventricular intracavitary injection as a cell delivery approach in infarcted mice. | Molecular and Cellular Biochemistry | 2021 |
| 32937086 | After the storm: an objective appraisal of the efficacy of c-kit+ cardiac progenitor cells in preclinical models of heart disease. | Canadian Journal of Physiology and Pharmacology | 2021 |
| 33118146 | Comparison of One and Three Intraventricular Injections of Cardiac Progenitor Cells in a Murine Model of Chronic Ischemic Cardiomyopathy. | Stem Cell Reviews and Reports | 2021 |
| 33389682 | Physiological Oxygen Tension Enhances Competence and Functional Properties of Murine Cardiac Mesenchymal Cells. | Stem Cell Reviews and Reports | 2021 |
| 34400167 | Exercise-induced late preconditioning in mice is triggered by eNOS-dependent generation of nitric oxide and activation of PKCε and is mediated by increased iNOS activity. | International Journal of Cardiology | 2021 |
| 32901075 | Administration of cardiac mesenchymal cells modulates innate immunity in the acute phase of myocardial infarction in mice. | Scientific Reports | 2020 |
| 32442025 | Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarction. | American Journal of Physiology - Heart and Circulatory Physiology | 2020 |
| 32515247 | Cardiospecific Overexpression of ATPGD1 (Carnosine Synthase) Increases Histidine Dipeptide Levels and Prevents Myocardial Ischemia Reperfusion Injury. | Journal of the American Heart Association | 2020 |
| 30735434 | TRPA1 channel contributes to myocardial ischemia-reperfusion injury. | American Journal of Physiology - Heart and Circulatory Physiology | 2019 |
| 31278427 | Inducible cardiac-specific overexpression of cyclooxygenase-2 (COX-2) confers resistance to ischemia/reperfusion injury. | Basic Research in Cardiology | 2019 |
| 30902920 | Correction: The heme oxygenase 1 inducer (CoPP) protects human cardiac stem cells against apoptosis through activation of the extracellular signal-related kinase (ERK)/NRF2 signaling pathway and cytokine release. | Journal of Biological Chemistry | 2019 |
| 30353243 | Cardiac mesenchymal cells from diabetic mice are ineffective for cell therapy-mediated myocardial repair. | Basic Research in Cardiology | 2018 |
| 28210871 | Repeated doses of cardiac mesenchymal cells are therapeutically superior to a single dose in mice with old myocardial infarction. | Basic Research in Cardiology | 2017 |
| 28385312 | Myocardial Reparative Properties of Cardiac Mesenchymal Cells Isolated on the Basis of Adherence. | Journal of the American College of Cardiology | 2017 |
| 26169370 | Genetic Deficiency of Glutathione S-Transferase P Increases Myocardial Sensitivity to Ischemia-Reperfusion Injury. | Circulation Research | 2015 |
| 25499773 | The NHLBI-sponsored Consortium for preclinicAl assESsment of cARdioprotective therapies (CAESAR): a new paradigm for rigorous, accurate, and reproducible evaluation of putative infarct-sparing interventions in mice, rabbits, and pigs. | Circulation Research | 2015 |
| 26299779 | Preconditioning Human Cardiac Stem Cells with an HO-1 Inducer Exerts Beneficial Effects After Cell Transplantation in the Infarcted Murine Heart. | Stem Cells | 2015 |
| 25151953 | Endoplasmic reticulum stress-dependent activation of ATF3 mediates the late phase of ischemic preconditioning. | Journal of Molecular and Cellular Cardiology | 2014 |
| 24806457 | c-kit+ Cardiac stem cells alleviate post-myocardial infarction left ventricular dysfunction despite poor engraftment and negligible retention in the recipient heart. | PLoS ONE | 2014 |
| 23549981 | A highly sensitive and accurate method to quantify absolute numbers of c-kit+ cardiac stem cells following transplantation in mice. | Basic Research in Cardiology | 2013 |
| 22119801 | Carbon monoxide induces a late preconditioning-mimetic cardioprotective and antiapoptotic milieu in the myocardium. | Journal of Molecular and Cellular Cardiology | 2012 |
| 22351077 | Identification of inducible nitric oxide synthase in peripheral blood cells as a mediator of myocardial ischemia/reperfusion injury. | Basic Research in Cardiology | 2012 |
| 22844439 | The COX-2/PGI2 receptor axis plays an obligatory role in mediating the cardioprotection conferred by the late phase of ischemic preconditioning. | PLoS ONE | 2012 |
| 22879597 | The heme oxygenase 1 inducer (CoPP) protects human cardiac stem cells against apoptosis through activation of the extracellular signal-regulated kinase (ERK)/NRF2 signaling pathway and cytokine release. | Journal of Biological Chemistry | 2012 |
| 22864681 | Genetic background, gender, age, body temperature, and arterial blood pH have a major impact on myocardial infarct size in the mouse and need to be carefully measured and/or taken into account: results of a comprehensive analysis of determinants of infarc | Basic Research in Cardiology | 2012 |
| 21779912 | Gene transfer as a strategy to achieve permanent cardioprotection I: rAAV-mediated gene therapy with inducible nitric oxide synthase limits infarct size 1Â year later without adverse functional consequences. | Basic Research in Cardiology | 2011 |
| 21516491 | Intracoronary administration of cardiac stem cells in mice: a new, improved technique for cell therapy in murine models. | Basic Research in Cardiology | 2011 |
| 21223971 | A murine model of inducible, cardiac-specific deletion of STAT3: its use to determine the role of STAT3 in the upregulation of cardioprotective proteins by ischemic preconditioning. | Journal of Molecular and Cellular Cardiology | 2011 |
| 20629987 | Transplantation of expanded bone marrow-derived very small embryonic-like stem cells (VSEL-SCs) improves left ventricular function and remodelling after myocardial infarction. | Journal of Cellular and Molecular Medicine | 2011 |
| 21785893 | Gene transfer as a strategy to achieve permanent cardioprotection II: rAAV-mediated gene therapy with heme oxygenase-1 limits infarct size 1Â year later without adverse functional consequences. | Basic Research in Cardiology | 2011 |
| 20081317 | Cardiac progenitor cells and bone marrow-derived very small embryonic-like stem cells for cardiac repair after myocardial infarction. | Circulation Journal | 2010 |
| 19616005 | The beneficial effects of postinfarct cytokine combination therapy are sustained during long-term follow-up. | Journal of Molecular and Cellular Cardiology | 2009 |
| 19752329 | Gene transfer of inducible nitric oxide synthase affords cardioprotection by upregulating heme oxygenase-1 via a nuclear factor-{kappa}B-dependent pathway. | Circulation | 2009 |
| 18936326 | Cardiac myocyte-specific expression of inducible nitric oxide synthase protects against ischemia/reperfusion injury by preventing mitochondrial permeability transition. | Circulation | 2008 |
| 18468618 | Acrolein consumption exacerbates myocardial ischemic injury and blocks nitric oxide-induced PKCepsilon signaling and cardioprotection. | Journal of Molecular and Cellular Cardiology | 2008 |
| 18430437 | Bone marrow-derived pluripotent very small embryonic-like stem cells (VSELs) are mobilized after acute myocardial infarction. | Journal of Molecular and Cellular Cardiology | 2008 |
| 18291412 | Endothelial nitric oxide synthase is not necessary for the early phase of ischemic preconditioning in the mouse. | Journal of Molecular and Cellular Cardiology | 2008 |
| 18420834 | Transplantation of bone marrow-derived very small embryonic-like stem cells attenuates left ventricular dysfunction and remodeling after myocardial infarction. | Stem Cells | 2008 |
| 18824172 | The role of TNF-alpha receptors p55 and p75 in acute myocardial ischemia/reperfusion injury and late preconditioning. | Journal of Molecular and Cellular Cardiology | 2008 |
| 17873008 | CRYAB and HSPB2 deficiency alters cardiac metabolism and paradoxically confers protection against myocardial ischemia in aging mice. | American Journal of Physiology - Heart and Circulatory Physiology | 2007 |
| 17606840 | Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-activated protein kinase pSer-signal transducers and activators of transcription1/3 pathway. | Circulation | 2007 |
| 17646584 | Stromal cell derived factor-1 alpha confers protection against myocardial ischemia/reperfusion injury: role of the cardiac stromal cell derived factor-1 alpha CXCR4 axis. | Circulation | 2007 |
| 17785622 | Cardioprotection afforded by inducible nitric oxide synthase gene therapy is mediated by cyclooxygenase-2 via a nuclear factor-kappaB dependent pathway. | Circulation | 2007 |
| 17541820 | The late phase of preconditioning and its natural clinical application--gene therapy. | Heart Failure Reviews | 2007 |