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Author Details

Aviad Tsherniak
Broad Institute of Massachusetts Institute of Technology (MIT) and Harvard
2011
64
43
PMIDPaper TitleJournal TitlePublished Year
37237081Genome-scale functional genomics identify genes preferentially essential for multiple myeloma cells compared to other neoplasias.Nat Cancer2023
37612728Partial gene suppression improves identification of cancer vulnerabilities when CRISPR-Cas9 knockout is pan-lethal.Genome Biol2023
37237081Genome-scale functional genomics identify genes preferentially essential for multiple myeloma cells compared to other neoplasias.Nat Cancer2023
37612728Partial gene suppression improves identification of cancer vulnerabilities when CRISPR-Cas9 knockout is pan-lethal.Genome Biol2023
35085500Sparse dictionary learning recovers pleiotropy from human cell fitness screens.Cell Syst2022
35365656Author Correction: Identification of ADAR1 adenosine deaminase dependency in a subset of cancer cells.Nat Commun2022
35085500Sparse dictionary learning recovers pleiotropy from human cell fitness screens.Cell Syst2022
35365656Author Correction: Identification of ADAR1 adenosine deaminase dependency in a subset of cancer cells.Nat Commun2022
33417832An Embryonic Diapause-like Adaptation with Suppressed Myc Activity Enables Tumor Treatment Persistence.Cancer Cell2021
33753930A first-generation pediatric cancer dependency map.Nat Genet2021
34260938Synthetic Lethal Interaction between the ESCRT Paralog Enzymes VPS4A and VPS4B in Cancers Harboring Loss of Chromosome 18q or 16q.Cell Rep2021
34253920Genome-scale screens identify factors regulating tumor cell responses to natural killer cells.Nat Genet2021
33417832An Embryonic Diapause-like Adaptation with Suppressed Myc Activity Enables Tumor Treatment Persistence.Cancer Cell2021
33406420Functional Genomics Identify Distinct and Overlapping Genes Mediating Resistance to Different Classes of Heterobifunctional Degraders of Oncoproteins.Cell Rep2021
34930405Chronos: a cell population dynamics model of CRISPR experiments that improves inference of gene fitness effects.Genome Biol2021
33753930A first-generation pediatric cancer dependency map.Nat Genet2021
34930405Chronos: a cell population dynamics model of CRISPR experiments that improves inference of gene fitness effects.Genome Biol2021
34260938Synthetic Lethal Interaction between the ESCRT Paralog Enzymes VPS4A and VPS4B in Cancers Harboring Loss of Chromosome 18q or 16q.Cell Rep2021
34253920Genome-scale screens identify factors regulating tumor cell responses to natural killer cells.Nat Genet2021
33406420Functional Genomics Identify Distinct and Overlapping Genes Mediating Resistance to Different Classes of Heterobifunctional Degraders of Oncoproteins.Cell Rep2021
32613204Discovering the anti-cancer potential of non-oncology drugs by systematic viability profiling.Nat Cancer2020
33128048Pan-cancer single-cell RNA-seq identifies recurring programs of cellular heterogeneity.Nat Genet2020
32855387Multiplexed single-cell transcriptional response profiling to define cancer vulnerabilities and therapeutic mechanism of action.Nat Commun2020
32613204Discovering the anti-cancer potential of non-oncology drugs by systematic viability profiling.Nat Cancer2020
33326793Synthetic Lethal Interaction between the ESCRT Paralog Enzymes VPS4A and VPS4B in Cancers Harboring Loss of Chromosome 18q or 16q.Cell Rep2020
33326793Synthetic Lethal Interaction between the ESCRT Paralog Enzymes VPS4A and VPS4B in Cancers Harboring Loss of Chromosome 18q or 16q.Cell Rep2020
33128048Pan-cancer single-cell RNA-seq identifies recurring programs of cellular heterogeneity.Nat Genet2020
32855387Multiplexed single-cell transcriptional response profiling to define cancer vulnerabilities and therapeutic mechanism of action.Nat Commun2020
31160565Neuronal differentiation and cell-cycle programs mediate response to BET-bromodomain inhibition in MYC-driven medulloblastoma.Nat Commun2019
31577942Synthetic Lethal Interaction of SHOC2 Depletion with MEK Inhibition in RAS-Driven Cancers.Cell Rep2019
31862961Agreement between two large pan-cancer CRISPR-Cas9 gene dependency data sets.Nat Commun2019
31133756Mitochondrial metabolism promotes adaptation to proteotoxic stress.Nat Chem Biol2019
30755442MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors.Cancer Res2019
30971823WRN helicase is a synthetic lethal target in microsatellite unstable cancers.Nature2019
31164776Author Correction: Mitochondrial metabolism promotes adaptation to proteotoxic stress.Nat Chem Biol2019
31068703The landscape of cancer cell line metabolism.Nat Med2019
31068700Next-generation characterization of the Cancer Cell Line Encyclopedia.Nature2019
30898838Genome-Wide Interrogation of Human Cancers Identifies EGLN1 Dependency in Clear Cell Ovarian Cancers.Cancer Res2019
31160565Neuronal differentiation and cell-cycle programs mediate response to BET-bromodomain inhibition in MYC-driven medulloblastoma.Nat Commun2019
31461650Small-Molecule and CRISPR Screening Converge to Reveal Receptor Tyrosine Kinase Dependencies in Pediatric Rhabdoid Tumors.Cell Rep2019
31862961Agreement between two large pan-cancer CRISPR-Cas9 gene dependency data sets.Nat Commun2019
31577942Synthetic Lethal Interaction of SHOC2 Depletion with MEK Inhibition in RAS-Driven Cancers.Cell Rep2019
31461650Small-Molecule and CRISPR Screening Converge to Reveal Receptor Tyrosine Kinase Dependencies in Pediatric Rhabdoid Tumors.Cell Rep2019
30898838Genome-Wide Interrogation of Human Cancers Identifies EGLN1 Dependency in Clear Cell Ovarian Cancers.Cancer Res2019
31068703The landscape of cancer cell line metabolism.Nat Med2019
31068700Next-generation characterization of the Cancer Cell Line Encyclopedia.Nature2019
31164776Author Correction: Mitochondrial metabolism promotes adaptation to proteotoxic stress.Nat Chem Biol2019
30971823WRN helicase is a synthetic lethal target in microsatellite unstable cancers.Nature2019
30755442MDM2 and MDM4 Are Therapeutic Vulnerabilities in Malignant Rhabdoid Tumors.Cancer Res2019
31133756Mitochondrial metabolism promotes adaptation to proteotoxic stress.Nat Chem Biol2019
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Collaborators

Francisca Vazquez
Broad Institute of MIT and Harvard
Co-authored papers 49
William C Hahn
Broad Institute of MIT and Harvard, USA Dana-Farber Cancer Institute
Co-authored papers 45
David E Root
Broad Institute of Harvard and MIT
Co-authored papers 29
Todd R Golub
Broad Institute of MIT and Harvard
Co-authored papers 22
Jesse S Boehm
Broad Institute of Harvard and MIT
Co-authored papers 21
Barbara A Weir
Janssen Research and Development LLC
Co-authored papers 21
Glenn S Cowley
Broad Institute of Harvard and MIT
Co-authored papers 16
Matthew Meyerson
Broad Institute
Co-authored papers 14
Kimberly Stegmaier
Dana-Farber Cancer Institute
Co-authored papers 13
Levi A Garraway
Broad Institute of Harvard and MIT
Co-authored papers 11
Pablo Tamayo
University of California San Diego
Co-authored papers 10
Mahmoud Ghandi
Co-authored papers 10
Jill P Mesirov
University of California San Diego
Co-authored papers 10
Andrew D Cherniack
Dana-Farber Cancer Institute
Co-authored papers 9
Charles W M Roberts
Co-authored papers 8
Brenton R Paolella
Co-authored papers 8
Rameen Beroukhim
Co-authored papers 8
Guillaume Kugener
Keck School of Medicine of the University of California los angeles
Co-authored papers 8
Gad Getz
Broad Institute of the Massachusetts Institute of Technology and Harvard
Co-authored papers 7
Gregory V Kryukov
Co-authored papers 6
Stuart L Schreiber
Broad Institute
Co-authored papers 5
Joseph Rosenbluh
Co-authored papers 5
Travis I Zack
Co-authored papers 5
Gabriela Alexe
Dana-Farber Cancer Institute
Co-authored papers 5
Matthew G Rees
Co-authored papers 5
Federica Piccioni
Co-authored papers 5
James E Bradner
Co-authored papers 5
Paul A Clemons
Broad Institute
Co-authored papers 4
Alykhan F Shamji
Center for the Science of Therapeutics, Broad Institute
Co-authored papers 4
Bang Wong
Co-authored papers 4
Release 1.2