Author details

Full Name

Lewis Kaufman

Affiliation

ORCID

Career Start Year

2004

Papers

H Index

Expertise

CM4AI Collaborator

PMID	Paper Title	Journal Title	Published Year
36523164	SARS-CoV-2 viral protein ORF3A injures renal tubules by interacting with TRIM59 to induce STAT3 activation.	Mol Ther	2023
34473647	AMPK mediates regulation of glomerular volume and podocyte survival.	JCI Insight	2021
33998601	DACH1 protects podocytes from experimental diabetic injury and modulates PTIP-H3K4Me3 activity.	J Clin Invest	2021
33998598	Transcriptome-wide association analysis identifies DACH1 as a kidney disease risk gene that contributes to fibrosis.	J Clin Invest	2021
33127607	Outcomes of Patients on Maintenance Dialysis Hospitalized with COVID-19.	Clin J Am Soc Nephrol	2021
32511564	Acute Kidney Injury in Hospitalized Patients with COVID-19.	medRxiv	2020
31171376	Disruption of MAGI2-RapGEF2-Rap1 signaling contributes to podocyte dysfunction in congenital nephrotic syndrome caused by mutations in MAGI2.	Kidney Int	2019
29773874	Mutations in six nephrosis genes delineate a pathogenic pathway amenable to treatment.	Nat Commun	2018
30341149	SHROOM3-FYN Interaction Regulates Nephrin Phosphorylation and Affects Albuminuria in Allografts.	Journal of the American Society of Nephrology : JASN	2018
28982981	The Hippo pathway regulator KIBRA promotes podocyte injury by inhibiting YAP signaling and disrupting actin cytoskeletal dynamics.	Journal of Biological Chemistry	2017
27707879	MAGI-1 Interacts with Nephrin to Maintain Slit Diaphragm Structure through Enhanced Rap1 Activation in Podocytes.	J Biol Chem	2016
24642466	Podocyte-specific RAP1GAP expression contributes to focal segmental glomerulosclerosis-associated glomerular injury.	J Clin Invest	2014
24449909	Essential role of poly(ADP-ribosyl)ation in cocaine action.	Proceedings of the National Academy of Sciences of the United States of America	2014
23325078	Expression of HIV transgene aggravates kidney injury in diabetic mice.	Kidney Int	2013
23842188	Podocyte-specific deletion of signal transducer and activator of transcription 3 attenuates nephrotoxic serum-induced glomerulonephritis.	Kidney International	2013
23343908	Deletion of podocyte STAT3 mitigates the entire spectrum of HIV-1-associated nephropathy.	AIDS	2013
21150871	Role of the retinoic acid receptor-Î± in HIV-associated nephropathy.	Kidney Int	2011
20562105	Up-regulation of the homophilic adhesion molecule sidekick-1 in podocytes contributes to glomerulosclerosis.	J Biol Chem	2010
20005487	The pathogenesis of HIV-associated nephropathy.	Advances in Chronic Kidney Disease	2010
17307840	The homophilic adhesion molecule sidekick-1 contributes to augmented podocyte aggregation in HIV-associated nephropathy.	FASEB Journal	2007
16495380	Role of ubiquitin-like protein FAT10 in epithelial apoptosis in renal disease.	J Am Soc Nephrol	2006
16763488	HIV-1 infection initiates an inflammatory cascade in human renal tubular epithelial cells.	Journal of acquired immune deficiency syndromes (1999)	2006
15703275	Definition of the critical domains required for homophilic targeting of mouse sidekick molecules.	FASEB Journal	2005
15213259	Sidekick-1 is upregulated in glomeruli in HIV-associated nephropathy.	Journal of the American Society of Nephrology : JASN	2004