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Author Details
Full Name
Matthew S Zabriskie
Affiliation
University of Utah
ORCID
Career Start Year
2011
Papers
23
H Index
15
Expertise
CM4AI Collaborator
PMID
Paper Title
Journal Title
Published Year
35306920
Spatially resolved transcriptomics for evaluation of intracranial vessels in a rabbit model: Proof of concept.
Interv Neuroradiol
2023
36847368
Real-Time Monitoring and Modulation of Blood Pressure in a Rabbit Model of Ischemic Stroke.
J Vis Exp
2023
34780648
MS4A3 promotes differentiation in chronic myeloid leukemia by enhancing common β-chain cytokine receptor endocytosis.
Blood
2022
34027418
SIRT5 IS A DRUGGABLE METABOLIC VULNERABILITY IN ACUTE MYELOID LEUKEMIA.
Blood Cancer Discov
2021
32613180
Apolipoprotein E knockout rabbit model of intracranial atherosclerotic disease.
Animal Model Exp Med
2020
31543464
Combining the Allosteric Inhibitor Asciminib with Ponatinib Suppresses Emergence of and Restores Efficacy against Highly Resistant BCR-ABL1 Mutants.
Cancer Cell
2019
29284681
A novel <i>AGGF1-PDGFRb</i> fusion in pediatric T-cell acute lymphoblastic leukemia.
Haematologica
2018
28819281
Mechanisms of resistance to the BCR-ABL1 allosteric inhibitor asciminib.
Leukemia
2017
28465516
Combined STAT3 and BCR-ABL1 inhibition induces synthetic lethality in therapy-resistant chronic myeloid leukemia.
Leukemia
2017
26582647
Extreme mutational selectivity of axitinib limits its potential use as a targeted therapeutic for BCR-ABL1-positive leukemia.
Leukemia
2016
26673800
A Novel Crizotinib-Resistant Solvent-Front Mutation Responsive to Cabozantinib Therapy in a Patient with ROS1-Rearranged Lung Cancer.
Clin Cancer Res
2016
26648538
Age-related mutations and chronic myelomonocytic leukemia.
Leukemia
2016
25134459
Combined STAT3 and BCR-ABL1 inhibition induces synthetic lethality in therapy-resistant chronic myeloid leukemia.
Leukemia
2015
25912019
Limited efficacy of BMS-911543 in a murine model of Janus kinase 2 V617F myeloproliferative neoplasm.
Exp Hematol
2015
25676420
Radotinib is an effective inhibitor of native and kinase domain-mutant BCR-ABL1.
Leukemia
2015
25721898
A coiled-coil mimetic intercepts BCR-ABL1 dimerization in native and kinase-mutant chronic myeloid leukemia.
Leukemia
2015
25573989
shRNA library screening identifies nucleocytoplasmic transport as a mediator of BCR-ABL1 kinase-independent resistance.
Blood
2015
25132497
BCR-ABL1 compound mutations combining key kinase domain positions confer clinical resistance to ponatinib in Ph chromosome-positive leukemia.
Cancer Cell
2014
23223358
BCR-ABL1 compound mutations in tyrosine kinase inhibitor-resistant CML: frequency and clonal relationships.
Blood
2013
23576564
Threshold levels of ABL tyrosine kinase inhibitors retained in chronic myeloid leukemia cells determine their commitment to apoptosis.
Cancer Res
2013
22825216
Pushing the limits of targeted therapy in chronic myeloid leukaemia.
Nat Rev Cancer
2012
21908430
The BCR-ABL35INS insertion/truncation mutant is kinase-inactive and does not contribute to tyrosine kinase inhibitor resistance in chronic myeloid leukemia.
Blood
2011
22169110
Nilotinib and MEK inhibitors induce synthetic lethality through paradoxical activation of RAF in drug-resistant chronic myeloid leukemia.
Cancer Cell
2011
1 - 23 of 23
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